Evidence map›Paper›PMID 31262823›Full record

ArticleProceedings of the National Academy of Sciences of the United States of America2019

Diversification of small RNA amplification mechanisms for targeting transposon-related sequences in ciliates.

Masatoshi Mutazono, Tomoko Noto, Kazufumi Mochizuki

Open access · bronzeAbstract read
In one paragraph

Article in Proceedings of the National Academy of Sciences of the United States of America, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.4field-weighted citation impact, top 38% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 11 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Programmed genome rearrangements in ciliates.Cellular and molecular life sciences : CMLS · 2020
    Review
  5. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Masatoshi MutazonoInstitute of Human Genetics, CNRS, University of Montpellier, 34090 Montpellier, France.
Tomoko NotoInstitute of Human Genetics, CNRS, University of Montpellier, 34090 Montpellier, France.
Kazufumi MochizukiInstitute of Human Genetics, CNRS, University of Montpellier, 34090 Montpellier, France kazufumi.mochizuki@igh.cnrs.fr.ORCID 0000-0001-7987-9852
Centre National de la Recherche Scientifique · FR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The silencing of repetitive transposable elements (TEs) is ensured by signal amplification of the initial small RNA trigger, which occurs at distinct steps of TE silencing in different eukaryotes. How such a variety of secondary small RNA biogenesis mechanisms has evolved has not been thoroughly elucidated. Ciliated protozoa perform small RNA-directed programmed DNA elimination of thousands of TE-related internal eliminated sequences (IESs) in the newly developed somatic nucleus. In the ciliate

Indexed as

Cell NucleusDNA End-Joining RepairDNA, ProtozoanDNA Transposable ElementsGene AmplificationGenome, ProtozoanGenomic InstabilityParameciumRNA, ProtozoanRNA, Small NuclearTetrahymenaDNA, ProtozoanDNA Transposable ElementsRNA, ProtozoanRNA, Small NuclearciliateDNA eliminationsmall RNATetrahymenatransposon silencing

Identifiers

PMID31262823
PMCPMC6642352
OpenAlexW2955803744

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.