Evidence map›Paper›PMID 31236120›Full record

ArticleCellular & molecular biology letters2019

Dp71 depleted HBE cells displayed increased DNA damage and apoptosis induced by H

Sichuang Tan, Shuai Zhao, Xuefei Xiao, Lan Xiao, Jinliang Xie, Sipin Tan

Open access · goldAbstract read
In one paragraph

Article in Cellular & molecular biology letters, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.2field-weighted citation impact, top 48% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 4 citations in OpenAlex.

  1. Article
  2. Review
  3. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

Sichuang Tan2Department of Thoracic Surgery, the Second Xiangya Hospital, Central South University, 139 Ren-min Road, Changsha, Hunan Province 410011 People's Republic of China.
Shuai Zhao2Department of Thoracic Surgery, the Second Xiangya Hospital, Central South University, 139 Ren-min Road, Changsha, Hunan Province 410011 People's Republic of China.
Xuefei Xiao5Department of Emergency and Critical Care Medicine, the Third Xiangya Hospital, Central South University, Changsha, Hunan Province People's Republic of China.
Lan Xiao4Department of Traditional Chinese Medicine, the Third Xiangya Hospital, Central South University, Changsha, Hunan Province People's Republic of China.
Jinliang Xie3Center of Transplant Surgery, Xiangya Hospital, Central South University, Changsha, Hunan Province 410008 People's Republic of China.
Sipin Tan1Key Laboratory of Sepsis Translational Medicine of Hunan, Department of Pathophysiology, Xiangya School of Medicine, Central South University, Changsha, Hunan Province 410008 People's Republic of China.ORCID 0000-0002-4271-6256
Central South University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Human bronchial epithelium (HBE)-Dp71 anti-sense(AS)cells with stably transfected Dp71 siRNA plasmids were prepared for further exploration of Dp71 biological traits in cells other than PC12. HBE-Dp71AS cells displayed increased DNA damage induced by H

Indexed as

ApoptosisDNA DamageOxidative StressCell LineDNADNA RepairDystrophinEpithelial CellsFocal Adhesion Kinase 1Gene Expression RegulationHumansHydrogen PeroxideLamin Type BPhosphorylationProtein Processing, Post-TranslationalRad51 Recombinaseapo-dystrophin 1DNADystrophinFocal Adhesion Kinase 1Hydrogen PeroxideLamin Type BPTK2 protein, humanRAD51 protein, humanRad51 RecombinaseApoptosisDNA damageDp71FAKRAD51

Identifiers

PMID31236120
PMCPMC6580496
OpenAlexW2951754430

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.