Evidence map›Paper›PMID 31222984›Full record

ArticleJournal of Korean medical science2019

Associations between the

Dong Hwa Lee, Gun Woo Won, Yong Hee Lee, Eu Jeong Ku, Tae Keun Oh, Hyun Jeong Jeon

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Journal of Korean medical science, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.4field-weighted citation impact, top 31% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 3 citations in OpenAlex.

  1. Polymorphism in theDiabetes & vascular disease research
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 1 country.

Dong Hwa LeeDepartment of Internal Medicine, Chungbuk National University Hospital, Chungbuk National University College of Medicine, Cheongju, Korea.ORCID https://orcid.org/0000-0002-1552-3205
Gun Woo WonDepartment of Biochemistry, Chungbuk National University College of Medicine, Cheongju, Korea.ORCID https://orcid.org/0000-0001-9518-4639
Yong Hee LeeDepartment of Biochemistry, Chungbuk National University College of Medicine, Cheongju, Korea.ORCID https://orcid.org/0000-0002-5484-3981
Eu Jeong KuDepartment of Internal Medicine, Chungbuk National University Hospital, Chungbuk National University College of Medicine, Cheongju, Korea.ORCID https://orcid.org/0000-0001-5533-4989
Tae Keun OhDepartment of Internal Medicine, Chungbuk National University Hospital, Chungbuk National University College of Medicine, Cheongju, Korea.ORCID https://orcid.org/0000-0002-2998-9671
Hyun Jeong JeonDepartment of Internal Medicine, Chungbuk National University Hospital, Chungbuk National University College of Medicine, Cheongju, Korea. endoann@daum.net.ORCID https://orcid.org/0000-0003-2841-0688
Chungbuk National University Hospital · KRChungbuk National University · KR

Funding

Chungbuk National University Hospital
6 · The paper itself

Abstract

backgroundDiabetic nephropathy (DN) is the most serious microvascular complication of diabetes mellitus and is one of the leading causes of end stage renal failure. In previous studies, the contribution of genetic susceptibility to DN showed inconsistent results. In this study, we investigated the association between the solute carrier family 2 facilitated glucose transporter member 1 (SLC2A1)

methodsA total of 846 patients with T2DM (mean age, 61.3 ± 12.3 years; mean duration of T2DM, 10.3 ± 7.9 years; 55.3% men) who visited the Chungbuk National University Hospital were investigated. The

resultsThe AG + AA genotype showed a significantly higher risk of DN compared with the GG genotype in patients with a type 2 DM duration less than 10 years (12.4% vs. 4.2%;

conclusionThe SLC2A1

Indexed as

AgedAllelesDiabetes Mellitus, Type 2Diabetic NephropathiesFemaleGenotypeGlucose Transporter Type 1HumansLogistic ModelsMaleMiddle AgedPolymorphism, Single NucleotideRepublic of KoreaRisk FactorsGlucose Transporter Type 1SLC2A1 protein, humanDiabetes Mellitus, Type 2Diabetic NephropathiesHaeIIIPolymorphismSLC2A1

Identifiers

PMID31222984
PMCPMC6589405
OpenAlexW2951606981

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.