Evidence map›Paper›PMID 31218401›Full record

ArticleEuropean journal of pediatrics2019

Genotype and phenotype analysis of a cohort of patients with congenital hyperinsulinism based on DOPA-PET CT scanning.

Jinwen Ni, Jingjie Ge, Miaoying Zhang, Khalid Hussain, Yihui Guan, Ruoqian Cheng, Li Xi, Zhangqian Zheng, Shuhua Ren, Feihong Luo

Open access · hybridAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in European journal of pediatrics, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
2.1field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 18 citations in OpenAlex.

  1. Article
  2. Review
  3. Congenital Hyperinsulinism Caused by Mutations inInternational journal of molecular sciences · 2024
    Article
  4. Article
  5. Review
  6. Article
  7. Article
  8. Article
  9. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 4 institutions in 2 countries.

Jinwen NiDepartment of Endocrinology and Inborn Metabolic Diseases, Children's Hospital of Fudan University, 399 Wanyuan Road, Shanghai, 201102, China.
Jingjie GePET CT Center, Division of Nuclear Medicine, Huashan Hospital, Fudan University, 518 East Wuzhong Road, Shanghai, 200235, China.
Miaoying ZhangDepartment of Endocrinology and Inborn Metabolic Diseases, Children's Hospital of Fudan University, 399 Wanyuan Road, Shanghai, 201102, China.
Khalid HussainDepartment of Pediatrics, Division of Endocrinology, Sidra Medicine OPC, C6-340 PO Box 26999, Al Luqta Street Education City North Campus, Doha, Qatar.
Yihui GuanPET CT Center, Division of Nuclear Medicine, Huashan Hospital, Fudan University, 518 East Wuzhong Road, Shanghai, 200235, China.
Ruoqian ChengDepartment of Endocrinology and Inborn Metabolic Diseases, Children's Hospital of Fudan University, 399 Wanyuan Road, Shanghai, 201102, China.
Li XiDepartment of Endocrinology and Inborn Metabolic Diseases, Children's Hospital of Fudan University, 399 Wanyuan Road, Shanghai, 201102, China.
Zhangqian ZhengDepartment of Endocrinology and Inborn Metabolic Diseases, Children's Hospital of Fudan University, 399 Wanyuan Road, Shanghai, 201102, China.
Shuhua RenPET CT Center, Division of Nuclear Medicine, Huashan Hospital, Fudan University, 518 East Wuzhong Road, Shanghai, 200235, China.
Feihong LuoDepartment of Endocrinology and Inborn Metabolic Diseases, Children's Hospital of Fudan University, 399 Wanyuan Road, Shanghai, 201102, China. luofh@fudan.edu.cn.
Children's Hospital of Fudan University · CNFudan University · CNHuashan Hospital · CNQatar Airways (Qatar) · QA

Funding

Shanghai Science and Technology Committee 15411951700
6 · The paper itself

Abstract

Congenital hyperinsulinism (CHI) is a clinically, genetically, and morphologically heterogeneous disorder.

Indexed as

GenotypePhenotypeAsian PeopleChildChild, PreschoolChinaCongenital HyperinsulinismDihydroxyphenylalanineFemaleGenetic MarkersGenetic TestingHumansInfantInfant, NewbornKcnj11 ChannelMaleABCC8 protein, humanDihydroxyphenylalaninefluorodopa F 18Genetic MarkersKcnj11 ChannelPotassium Channels, Inwardly RectifyingRadiopharmaceuticalsSulfonylurea ReceptorsCongenital hyperinsulinismGenetic association studiesHyperinsulinemiaHypoglycemiaMutation

Identifiers

PMID31218401
PMCPMC6647509
OpenAlexW2953268729

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.