Evidence map›Paper›PMID 31168358›Full record

ReviewF1000Research2019

Immunology of membranous nephropathy.

Shin'ichi Akiyama, Enyu Imai, Shoichi Maruyama

Abstract readReview
In one paragraph

Review in F1000Research, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed.

  1. Review
  2. Article
  3. Membranous Nephropathy.Journal of clinical medicine · 2025
    Review
  4. Article
  5. Review
  6. Article
  7. Article
  8. Review
  9. [Pharmacogenetic testing improves treatment responses in patients with PLA2R-related membranous nephropathy].Nan fang yi ke da xue xue bao = Journal of Southern Medical University · 2023
    Article
  10. Article
  11. Article
  12. Review
  13. Article
  14. Article
  15. Review
  16. Article
  17. Complement and Complement Targeting Therapies in Glomerular Diseases.International journal of molecular sciences · 2019
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Shin'ichi AkiyamaDivision of Nephrology, Department of Internal Medicine, Nagoya University Graduate School of Medicine, Nagoya, Japan.ORCID 0000-0003-4313-2911
Enyu ImaiNakayamadera Imai Clinic, Takarazuka, Hyōgo, Japan.
Shoichi MaruyamaDivision of Nephrology, Department of Internal Medicine, Nagoya University Graduate School of Medicine, Nagoya, Japan.ORCID 0000-0002-8858-632X

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Accounting for about 20 to 50% of cases of primary nephrotic syndrome, membranous nephropathy (MN) is the leading cause of nephrotic syndrome in adults. A rat model created nearly 60 years ago to research the primary MN disorder, Heymann nephritis, has provided us with a plethora of important information. Recently, our knowledge about MN has dramatically progressed. Heymann nephritis and human MN are now known to share a high degree of similarity in pathogenesis. This review summarizes our current understanding of MN pathogenesis while focusing particularly on the immunological aspects.

Indexed as

AnimalsDisease Models, AnimalGlomerulonephritis, MembranousHumansRatsEpitope spreadingHeymann nephritisMembranous nephropathyNEPPLA2RpodocyteTHSD7A

Identifiers

PMID31168358
PMCPMC6537914

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.