Evidence map›Paper›PMID 31167495›Full record

ArticleCells2019

Low VDAC1 Expression Is Associated with an Aggressive Phenotype and Reduced Overall Patient Survival in Cholangiocellular Carcinoma.

René Günther Feichtinger, Daniel Neureiter, Ralf Kemmerling, Johannes Adalbert Mayr, Tobias Kiesslich, Barbara Kofler

Open access · goldAbstract read
In one paragraph

Article in Cells, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
1.3field-weighted citation impact, top 20% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 11 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. The expression ofFrontiers in endocrinology · 2024
    Article
  6. Review
  7. Article
  8. Article
  9. Article
  10. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

René Günther FeichtingerResearch Program for Receptor Biochemistry and Tumor Metabolism, Department of Pediatrics, University Hospital Salzburg of the Paracelsus Medical University, 5020 Salzburg, Austria. r.feichtinger@salk.at.
Daniel NeureiterInstitute of Pathology, University Hospital Salzburg of the Paracelsus Medical University, 5020 Salzburg, Austria. d.neureiter@salk.at.
Ralf KemmerlingInstitute of Pathology, University Hospital Salzburg of the Paracelsus Medical University, 5020 Salzburg, Austria. ralf.kemmerling@web.de.
Johannes Adalbert MayrDepartment of Pediatrics, University Hospital Salzburg of the Paracelsus Medical University, 5020 Salzburg, Austria. h.mayr@salk.at.
Tobias KiesslichLaboratory for Tumor Biology and Experimental Therapies (TREAT), Institute of Physiology and Pathophysiology, Paracelsus Medical University, 5020 Salzburg, Austria. tobias.kiesslich@pmu.ac.at.
Barbara KoflerResearch Program for Receptor Biochemistry and Tumor Metabolism, Department of Pediatrics, University Hospital Salzburg of the Paracelsus Medical University, 5020 Salzburg, Austria. b.kofler@salk.at.
Paracelsus Medical University · AT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

PubMed holds no abstract for this paper.

Indexed as

Bile Duct NeoplasmsCholangiocarcinomaElectron Transport Complex IEnergy MetabolismFemaleHumansKaplan-Meier EstimateMaleMitochondriaNeoplasm StagingOxidative PhosphorylationPhenotypeVoltage-Dependent Anion Channel 1Electron Transport Complex INDUFS4 protein, humanVDAC1 protein, humanVoltage-Dependent Anion Channel 1cholangiocellular carcinomaenergy metabolismmitochondriaoxidative phosphorylation

Identifiers

PMID31167495
PMCPMC6627691
OpenAlexW2948317085

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.