ArticleAmerican journal of respiratory and critical care medicine2019
The Transferrin Receptor CD71 Delineates Functionally Distinct Airway Macrophage Subsets during Idiopathic Pulmonary Fibrosis.
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Article in American journal of respiratory and critical care medicine, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 82 papers.
0numbers the graph read from it
0cells of the map it votes in
82citing papers in PubMed
11.7field-weighted citation impact, top 1% of its field
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
82 citing papers in PubMed, 108 citations in OpenAlex.
- Exploring Ferroptosis: A Key Mechanism in Respiratory System Diseases and its Therapeutic Potential.Applied biochemistry and biotechnology · 2026Review
- Cellular Senescence: Emerging Therapeutic Target for Idiopathic Pulmonary Fibrosis Pathogenic Mechanisms and Therapeutic Strategies.Biomolecules & therapeutics · 2026Review
- Iron and the lung: emerging roles of iron metabolism in pulmonary disease pathogenesis and therapy.European respiratory review : an official journal of the European Respiratory Society · 2026Review
- Hallmarks of the ageing lung: 10 years later.The European respiratory journal · 2026Review
- Review
- Single-cell assessment of iron content in primary human T cells using laser ablation inductively coupled plasma mass spectrometry.Cell reports methods · 2026Article
- Profibrotic macrophage populations and cell communications in pulmonary fibrosis.Journal of physiology and biochemistry · 2026Review
- Interaction ofiScience · 2026Article
- Downregulation of SLC40A1 leads to iron accumulation in fibrotic lung fibroblasts.Respiratory research · 2026Article
- Recent understanding of immunometabolic remodeling in pulmonary macrophages: homeostasis, chronic respiratory diseases, and therapeutic targeting.Frontiers in pharmacology · 2026Review
- The impact of macrophage metabolism on idiopathic pulmonary fibrosis development and treatment prospectives.Frontiers in immunology · 2026Review
- Improved Iron Uptake and Metabolism Through Combined Heme and Non-Heme Iron Supplementation: An In Vitro Study.Biomedicines · 2025Article
- TMPRSS11E-mediated TFR1 cleavage influences IFN-γR2 internalization and the macrophage innate response.Communications biology · 2025Article
- NADJournal of neuroinflammation · 2025Article
- Iron metabolism dysregulation and ferroptosis: Emerging drivers in pulmonary fibrosis pathogenesis and therapy.Molecules and cells · 2025Review
- Loss of Iron Regulatory Protein 2 Protects Mice from Lung Inflammation during Bronchoconstriction.American journal of respiratory cell and molecular biology · 2025Article
- Fibrosis: cross-organ biology and pathways to development of innovative drugs.Nature reviews. Drug discovery · 2025Review
- Proteomics and Metabolomics Analyses Reveal a Dynamic Landscape of Coal Workers' Pneumoconiosis: An Insight into Disease Progression.Journal of proteome research · 2025Article
- PCR array analysis reveals a novel expression profile of ferroptosis-related genes in idiopathic pulmonary fibrosis.BMC pulmonary medicine · 2025Article
- Review
22 more citing papers are in PubMed but not listed here.
Corrections and comments
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- Commented on byCD712019
Authors and funding
13 authors at 2 institutions in 2 countries.
Sarah J Allden1 Inflammation, Repair, and Development Section, National Heart and Lung Institute, Faculty of Medicine, Imperial College, London, United Kingdom.
Patricia P Ogger1 Inflammation, Repair, and Development Section, National Heart and Lung Institute, Faculty of Medicine, Imperial College, London, United Kingdom.
Poonam Ghai1 Inflammation, Repair, and Development Section, National Heart and Lung Institute, Faculty of Medicine, Imperial College, London, United Kingdom.
Peter McErlean1 Inflammation, Repair, and Development Section, National Heart and Lung Institute, Faculty of Medicine, Imperial College, London, United Kingdom.ORCID 0000-0003-4199-3355
Richard Hewitt1 Inflammation, Repair, and Development Section, National Heart and Lung Institute, Faculty of Medicine, Imperial College, London, United Kingdom.ORCID 0000-0003-4294-0052
Richard Toshner1 Inflammation, Repair, and Development Section, National Heart and Lung Institute, Faculty of Medicine, Imperial College, London, United Kingdom.
Simone A Walker1 Inflammation, Repair, and Development Section, National Heart and Lung Institute, Faculty of Medicine, Imperial College, London, United Kingdom.
Peter Saunders3 NIHR Respiratory Biomedical Research Unit, Royal Brompton Hospital, London, United Kingdom.ORCID 0000-0001-9299-0299
Shaun Kingston3 NIHR Respiratory Biomedical Research Unit, Royal Brompton Hospital, London, United Kingdom.
Philip L Molyneaux1 Inflammation, Repair, and Development Section, National Heart and Lung Institute, Faculty of Medicine, Imperial College, London, United Kingdom.ORCID 0000-0003-1301-8800
Toby M Maher1 Inflammation, Repair, and Development Section, National Heart and Lung Institute, Faculty of Medicine, Imperial College, London, United Kingdom.ORCID 0000-0001-7192-9149
Clare M Lloyd1 Inflammation, Repair, and Development Section, National Heart and Lung Institute, Faculty of Medicine, Imperial College, London, United Kingdom.ORCID 0000-0001-8977-6726
Adam J Byrne1 Inflammation, Repair, and Development Section, National Heart and Lung Institute, Faculty of Medicine, Imperial College, London, United Kingdom.ORCID 0000-0003-2736-8174
Lung Institute · USRoyal Brompton Hospital · GB
Funding
Department of Health CS-2013-13-017HSRD VA CDA 13-017Wellcome TrustWellcome Trust 107059/Z/15/ZWellcome Trust 205949/Z/17/Z
Abstract
PubMed holds no abstract for this paper.
Indexed as
PhagocytosisAdultAgedAged, 80 and overAntigens, CDBronchoalveolar Lavage FluidCase-Control StudiesFemaleFlow CytometryGene Expression ProfilingHumansIdiopathic Pulmonary FibrosisIronMacrophages, AlveolarMaleMiddle AgedAntigens, CDCD71 antigenIronReceptors, TransferrinTransferrinairway macrophagesidiopathic pulmonary fibrosistransferrin receptor
Identifiers
What OpenQuestion holds
Textmetadata
LicenceCC BY
Read underepoch 390
Registered trials
None linked
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.