Evidence map›Paper›PMID 31046698›Full record

ArticleBMC nephrology2019

The atrial uremic cardiomyopathy regression in patients after kidney transplantation - the prospective echocardiographic study.

Tomasz Zapolski, Jacek Furmaga, Andrzej P Wysokiński, Anna Wysocka, Sławomir Rudzki, Andrzej Jaroszyński

Open access · goldAbstract read
In one paragraph

Article in BMC nephrology, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
1.3field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 21 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Article
  6. Factors associated with changes in echocardiographic parameters following kidney transplantation.Clinical research in cardiology : official journal of the German Cardiac Society · 2024
    Article
  7. Review
  8. Article
  9. Article
  10. Article
  11. Uraemic Cardiomyopathy: A Review of Current Literature.Clinical Medicine Insights. Cardiology · 2021
    Review
  12. Article
  13. Review
  14. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 1 country.

Tomasz ZapolskiChair and Department of Cardiology, Medical University of Lublin, Lublin, Poland. zapolia@wp.pl.ORCID 0000-0001-9808-8853
Jacek FurmagaDepartment of General and Transplant Surgery and Nutritional Treatment, Medical University of Lublin, Lublin, Poland.
Andrzej P WysokińskiChair and Department of Cardiology, Medical University of Lublin, Lublin, Poland.
Anna WysockaChair and Department of Cardiology, Medical University of Lublin, Lublin, Poland.
Sławomir RudzkiDepartment of General and Transplant Surgery and Nutritional Treatment, Medical University of Lublin, Lublin, Poland.
Andrzej JaroszyńskiDepartment of Nephrology, Jan Kochanowski University in Kielce, Kielce, Poland.
Medical University of Lublin · PLJan Kochanowski University · PL

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundIn patients with end stage renal disease (ESRD), left ventricular (LV) hypertrophy with impaired LV function, which is called uremic cardiomyopathy (UC) is often observed. The UC historically has been considered a contraindication for kidney transplantation (KTx). Currently, moderate LV dysfunction does not exclude the possibility of KTx. The amelioration of uremia after KTx improved cardiac function in patients with LV dysfunction. There is a little information on the function of the left atrium (LA) after the KTx procedure. There are no studies evaluating (LA) changes in patients with UC after KTx and determining the possibility of inhibiting the occurrence of LA unfavourable changes (remodelling) and even a possible LA recovery process (reverse remodelling) as a result of a successful KTx. The aim of the study was to assess the LA reverse remodelling in patients with ESRD undergoing KTx.

methodsThe study group consisted of 42 patients, aged 43.3 ± 12.6 followed for 36 months after a deceased donor KTx. The patients were studied at five time points: 1, 3, 6, 12 and 36 months after KTx. In all patients transthoracic echocardiography was performed in order to assess the following LA planimetric parameters: LA

resultsThe LAVI values were 34.63 ± 10.34 ml/m

conclusionsLA remodelling secondary to atrial uraemic cardiomyopathy is an example of complex cardiomyopathy with elements characteristic of both congestive and infiltrative cardiomyopathy. Early LAVI reduction post KTx mostly depends on changed haemodynamic conditions, whereas the main reason for further decrease of LAVI values is related to resolution of uraemic toxaemia.

Indexed as

Kidney TransplantationAdultAtrial RemodelingCreatinineEchocardiographyFemaleGlomerular Filtration RateHeart AtriaHumansHypertrophy, Left VentricularKidney Failure, ChronicMaleMyocardial ContractionOrgan SizeProspective StudiesRegression AnalysisCreatinineAtrial uraemic cardiomyopathyEnd-stage renal diseaseKidney transplantationLeft atrium volume index

Identifiers

PMID31046698
PMCPMC6498637
OpenAlexW2947645924

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LicenceCC BY
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.