O-GlcNAc Transferase Suppresses Inflammation and Necroptosis by Targeting Receptor-Interacting Serine/Threonine-Protein Kinase 3.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

29 citing papers in PubMed.

1. Arginine Di-methylation of RIP3 safeguards necroptosis for intestinal homeostasis.Cell death and differentiation · 2026
   Article
2. O-GlcNAcylation regulates microglial neuroinflammation in Parkinson's disease.NPJ Parkinson's disease · 2026
   Article
3. Epigenomic and Metabolic Interplay in the Development of Metastatic Brain Tumors.Oncology research · 2026
   Review
4. Toll-like receptors in innate immunity and inflammation: from fundamental biology to clinic insights.Immunity & inflammation · 2026
   Review
5. Crosstalk of regulated cell death pathways in sepsis-associated acute kidney injury: implications for therapy.Frontiers in cell and developmental biology · 2026
   Review
6. Pattern-Recognition Receptors and Immunometabolic Reprogramming: What We Know and What to Explore.Journal of innate immunity · 2024
   Review
7. The role of O-GlcNAcylation in innate immunity and inflammation.Journal of molecular cell biology · 2023
   Review
8. Necroptosis Related Genes Predict Prognosis and Therapeutic Potential in Gastric Cancer.Biomolecules · 2023
   Article
9. Frontiers in immunology · 2023
   Article
10. High-expression of the innate-immune related gene UNC93B1 predicts inferior outcomes in acute myeloid leukemia.Frontiers in genetics · 2023
    Article
11. Mechanisms of TNF-independent RIPK3-mediated cell death.The Biochemical journal · 2022
    Review
12. Ubiquitylation of RIPK3 beyond-the-RHIM can limit RIPK3 activity and cell death.iScience · 2022
    Article
13. The interaction of O-GlcNAc-modified NLRX1 and IKK-α modulates IL-1β expression in M1 macrophages.In vitro cellular & developmental biology. Animal · 2022
    Article
14. Roles of necroptosis in alcoholic liver disease and hepatic pathogenesis.Cell proliferation · 2022
    Review
15. O-GlcNAcylation regulation of cellular signaling in cancer.Cellular signalling · 2022
    Review
16. Inhibition of O-GlcNAcylation protects from Shiga toxin-mediated cell injury and lethality in host.EMBO molecular medicine · 2022
    Article
17. Machine learning identification of cuproptosis and necroptosis-associated molecular subtypes to aid in prognosis assessment and immunotherapy response prediction in low-grade glioma.Frontiers in genetics · 2022
    Article
18. Inhibition of O-GlcNAcylation Decreases the Cytotoxic Function of Natural Killer Cells.Frontiers in immunology · 2022
    Article
19. A Necroptosis-Related Prognostic Model of Uveal Melanoma Was Constructed by Single-Cell Sequencing Analysis and Weighted Co-Expression Network Analysis Based on Public Databases.Frontiers in immunology · 2022
    Article
20. NleB2 from enteropathogenic Escherichia coli is a novel arginine-glucose transferase effector.PLoS pathogens · 2021
    Article

Corrections and comments

• Erratum for
  O-GlcNAc Transferase Suppresses Inflammation and Necroptosis by Targeting Receptor-Interacting Serine/Threonine-Protein Kinase 3.2019

Authors and funding

19 authors.

Funding

Abstract

PubMed holds no abstract for this paper.