Evidence map›Paper›PMID 30965639›Full record

ArticleNon-coding RNA2019

Long Non-Coding RNA

Keisuke Hitachi, Masashi Nakatani, Kunihiro Tsuchida

Open access · goldAbstract read
In one paragraph

Article in Non-coding RNA, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
0.7field-weighted citation impact, top 37% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 16 citations in OpenAlex.

  1. Review
  2. Article
  3. Article
  4. Epigenetics of Skeletal Muscle Atrophy.International journal of molecular sciences · 2024
    Review
  5. Article
  6. Review
  7. Article
  8. Article
  9. Review
  10. Article
  11. Non-coding RNAs Shaping Muscle.Frontiers in cell and developmental biology · 2019
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Keisuke HitachiDivision for Therapies against Intractable Diseases, Institute for Comprehensive Medical Science (ICMS), Fujita Health University, Toyoake 470-1192, Japan. hkeisuke@fujita-hu.ac.jp.
Masashi NakataniDivision for Therapies against Intractable Diseases, Institute for Comprehensive Medical Science (ICMS), Fujita Health University, Toyoake 470-1192, Japan. nakatani@fujita-hu.ac.jp.
Kunihiro TsuchidaDivision for Therapies against Intractable Diseases, Institute for Comprehensive Medical Science (ICMS), Fujita Health University, Toyoake 470-1192, Japan. tsuchida@fujita-hu.ac.jp.
Fujita Health University · JP

Funding

Japan Society for the Promotion of Science 16K08599Japan Society for the Promotion of Science 17K08646Nakatomi Foundation 2017NCNP Intramural Research Grant (29-4) for Neurological and Psychiatric Disorders
6 · The paper itself

Abstract

Skeletal muscle is a highly plastic tissue and decreased skeletal muscle mass (muscle atrophy) results in deteriorated motor function and perturbed body homeostasis.

Indexed as

bone morphogenetic protein signalingdenervationpromoter-associated long non-coding RNARNA-seqskeletal muscle atrophy

Identifiers

PMID30965639
PMCPMC6631233
OpenAlexW2937552957

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.