Bilateral Destructive Hip Disease from Untreated Juvenile Idiopathic Arthritis.

Arash Calafi, Alton W Skaggs, Trevor J Shelton, Brian M Haus

Abstract readCase Reports

In one paragraph

Article in Case reports in orthopedics, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed

–field-weighted citation impact

1 · What the graph read from it

What it found

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2 · The registry

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3 · Its place in the literature

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0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

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PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Arash CalafiUniversity of Washington Department of Orthopaedics and Sports Medicine, 1959 N.E. Pacific St., Box 356500 Seattle, WA 98195, USA.

Alton W SkaggsSchool of Medicine at University of California at Davis, Sacramento, CA 95817, USA.ORCID 0000-0001-7752-4265

Trevor J SheltonDepartment of Orthopaedic Surgery, University of California at Davis, 4860 Y Street, Suite 3800, Sacramento, CA 95829, USA.ORCID 0000-0002-2032-3371

Brian M HausDepartment of Orthopaedic Surgery, University of California at Davis, 4860 Y Street, Suite 3800, Sacramento, CA 95829, USA.ORCID 0000-0002-0561-017X

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

We report a novel case of a pediatric patient with bilateral hip destruction from untreated Juvenile idiopathic arthritis (JIA). She was presented at the age of 9 with hip pain associated with bilateral acetabular dysplasia and a dislocated left femoral head. Only 1.5 years later, the patient developed complete destruction of the left femoral head and dislocated right femoral head. The authors have not identified literature describing a similar case report of bilateral femoral head destruction resulting from Persistent Oligoarticular JIA. Pediatric patients presenting with rapidly evolving destructive process should be evaluated for rheumatologic, infectious, and spinal etiologies.

Identifiers

PMID30963013

DOI10.1155/2019/4593129

PMCPMC6431357

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