Evidence map›Paper›PMID 30948355›Full record

ArticleBiology open2019

Distinct gene-selective roles for a network of core promoter factors in

Alexandre Neves, Robert N Eisenman

Abstract read
In one paragraph

Article in Biology open, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Article
  2. Epigenetic regulation of post-embryonic development.Current opinion in insect science · 2021
    Review
  3. The Core Promoter Is a Regulatory Hub for Developmental Gene Expression.Frontiers in cell and developmental biology · 2021
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Alexandre NevesBasic Sciences Division, Fred Hutchinson Cancer Research Center, Seattle, WA 98109-1024, USA aneves@fredhutch.org eisenman@fredhutch.org.ORCID http://orcid.org/0000-0003-2775-8447
Robert N EisenmanBasic Sciences Division, Fred Hutchinson Cancer Research Center, Seattle, WA 98109-1024, USA aneves@fredhutch.org eisenman@fredhutch.org.ORCID http://orcid.org/0000-0002-0274-9846

Funding

Resource Component: Acquisition, maintenance and distribution of Drosophila stocksP40OD018537 · OD · TRUSTEES OF INDIANA UNIVERSITY · PI Annette L. Parks · 2014 to 2026
$13.5M
Drosophila Transgenic RNAi Resource ProjectR01GM084947 · NIGMS · HARVARD MEDICAL SCHOOL · PI PERRIMON, NORBERT · 2008 to 2019
$9.8M
MYC ONCOPROTEIN FUNCTIONR37CA057138 · NCI · FRED HUTCHINSON CANCER RESEARCH CENTER · PI EISENMAN, ROBERT NEIL · 2003 to 2013
$8.0M
Chromosome Metabolism and Cancer Training GrantT32CA009657 · NCI · FRED HUTCHINSON CANCER RESEARCH CENTER · PI EISENMAN, ROBERT NEIL · 1991 to 2020
$7.6M
The MYC Transcription Factor Network and the Path to CancerR35CA231989 · NCI · FRED HUTCHINSON CANCER RESEARCH CENTER · PI EISENMAN, ROBERT NEIL · 2018 to 2024
$6.3M
MYC ONCOPROTEIN FUNCTIONR01CA057138 · NCI · FRED HUTCHINSON CANCER RESEARCH CENTER · PI EISENMAN, ROBERT NEIL · 1991 to 2018
$4.9M
Control of Neural Stem Cell Identity by Tafs and Trf2R21NS090037 · NINDS · FRED HUTCHINSON CANCER RESEARCH CENTER · PI EISENMAN, ROBERT NEIL · 2016 to 2017
$484k
NCI NIH HHS R01 CA057138NCI NIH HHS R35 CA231989NCI NIH HHS R37 CA057138NCI NIH HHS T32 CA009657NIGMS NIH HHS R01 GM084947NIH HHS P40 OD018537NINDS NIH HHS R21 NS090037
6 · The paper itself

Abstract

The transcriptional mechanisms that allow neural stem cells (NSC) to balance self-renewal with differentiation are not well understood. Employing an

Indexed as

Neural stem cellsTAFsTranscription

Identifiers

PMID30948355
PMCPMC6504003

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.