Evidence map›Paper›PMID 30919272›Full record

ArticleJournal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology2020

Correction to: Risk stratification with vasodilator stress SPECT myocardial perfusion imaging in patients with elevated cardiac biomarkers.

Shreyas Gowdar, Alan W Ahlberg, Mridula Rai, William H Perucki, Kevin D Felpel, John A Savino, Eric L Alter, Milena J Henzlova, W Lane Duvall

Abstract readPublished Erratum
PubMed Publisher
In one paragraph

Article in Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

9 authors.

Shreyas Gowdar *Hartford Hospital Division of Cardiology, Hartford Healthcare Heart and Vascular Institute, 80 Seymour Street, Hartford, CT, 06102, USA.
Alan W Ahlberg *Hartford Hospital Division of Cardiology, Hartford Healthcare Heart and Vascular Institute, 80 Seymour Street, Hartford, CT, 06102, USA.
Mridula RaiHartford Hospital Division of Cardiology, Hartford Healthcare Heart and Vascular Institute, 80 Seymour Street, Hartford, CT, 06102, USA.
William H PeruckiHartford Hospital Division of Cardiology, Hartford Healthcare Heart and Vascular Institute, 80 Seymour Street, Hartford, CT, 06102, USA.
Kevin D FelpelDepartment of Medicine, University of Connecticut School of Medicine, Farmington, USA.
John A SavinoMount Sinai Division of Cardiology, Mount Sinai Heart, Mount Sinai Medical Center, New York, NY, USA.
Eric L AlterMount Sinai Division of Cardiology, Mount Sinai Heart, Mount Sinai Medical Center, New York, NY, USA.
Milena J HenzlovaMount Sinai Division of Cardiology, Mount Sinai Heart, Mount Sinai Medical Center, New York, NY, USA.
W Lane DuvallHartford Hospital Division of Cardiology, Hartford Healthcare Heart and Vascular Institute, 80 Seymour Street, Hartford, CT, 06102, USA. lane.duvall@hhchealth.org.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The following error (highlighted in bold below) appears in the Results section of the Abstract: A total of 503 patients were followed for an average of 33.6 ± 16.2 months, with a mean age of 69.3 years; 53.7% male; and a majority (88.7%) of them undergoing vasodilator stress. A significant increase in all-cause mortality was seen based on the severity of TPD results for all vasodilators (P < .0001) and regadenoson (P < .0001). Similar prognostic ability was seen for all-cause (this should actually be cardiac) mortality. This association was maintained even after adjustment for cardiac risk factors, previous coronary disease, and troponin quartiles. MPI results (stress TPD and LVEF) added to traditional cardiac risk factors, and troponin values resulted in a significant incremental increase in the ability to predict all-cause and cardiac mortality, and stress TPD remained independently predictive for both all-cause and cardiac mortality in a multivariate model.

Identifiers

PMID30919272

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.