Evidence map›Paper›PMID 30891050›Full record

ArticleFrontiers in plant science2019

The MKK7-MPK6 MAP Kinase Module Is a Regulator of Meristem Quiescence or Active Growth in Arabidopsis.

Róbert Dóczi, Elizabeth Hatzimasoura, Sara Farahi Bilooei, Zaki Ahmad, Franck Anicet Ditengou, Enrique López-Juez, Klaus Palme, László Bögre

Open access · goldAbstract read
In one paragraph

Article in Frontiers in plant science, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
2.9field-weighted citation impact, top 9% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 18 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 3 countries.

Róbert DócziCentre for Systems and Synthetic Biology, School of Biological Sciences, Royal Holloway, University of London, Egham, United Kingdom.
Elizabeth HatzimasouraCentre for Systems and Synthetic Biology, School of Biological Sciences, Royal Holloway, University of London, Egham, United Kingdom.
Sara Farahi BilooeiCentre for Systems and Synthetic Biology, School of Biological Sciences, Royal Holloway, University of London, Egham, United Kingdom.
Zaki AhmadCentre for Systems and Synthetic Biology, School of Biological Sciences, Royal Holloway, University of London, Egham, United Kingdom.
Franck Anicet DitengouInstitute of Biology II, University of Freiburg, Freiburg im Breisgau, Germany.
Enrique López-JuezCentre for Systems and Synthetic Biology, School of Biological Sciences, Royal Holloway, University of London, Egham, United Kingdom.
Klaus PalmeInstitute of Biology II, University of Freiburg, Freiburg im Breisgau, Germany.
László BögreCentre for Systems and Synthetic Biology, School of Biological Sciences, Royal Holloway, University of London, Egham, United Kingdom.
Royal Holloway University of London · GBUniversity of Freiburg · DEHungarian Academy of Sciences · HU

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Plant growth flexibly adapts to environmental conditions. Growth initiation itself may be conditional to a suitable environment, while the most common response of plants to adverse conditions is growth inhibition. Most of our understanding about environmental growth inhibition comes from studies on various plant hormones, while less is known about the signaling mechanisms involved. The mitogen-activated protein kinase (MAPK) cascades are central signal transduction pathways in all eukaryotes and their roles in plant stress responses is well-established, while increasing evidence points to their involvement in hormonal and developmental processes. Here we show that the MKK7-MPK6 module is a suppressor of meristem activity using genetic approaches. Shoot apical meristem activation during light-induced de-etiolation is accelerated in

Indexed as

Arabidopsisde-etiolationMAP kinasemeristemsignaling

Identifiers

PMID30891050
PMCPMC6413535
OpenAlexW2918698921

What OpenQuestion holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.