Evidence map›Paper›PMID 30867511›Full record

ArticleScientific reports2019

PIP

Min-Jue Xie, Yasuyuki Ishikawa, Hideshi Yagi, Tokuichi Iguchi, Yuichiro Oka, Kazuki Kuroda, Keiko Iwata, Hiroshi Kiyonari, Shinji Matsuda, Hideo Matsuzaki and 3 more

Open access · goldAbstract read
In one paragraph

Article in Scientific reports, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed
1.6field-weighted citation impact, top 19% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed, 23 citations in OpenAlex.

  1. Article
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  5. Review
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  7. Review
  8. Sex-specific lipid dysregulation in theBrain communications · 2022
    Article
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  13. StandardizedAntioxidants (Basel, Switzerland) · 2020
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors at 3 institutions in 1 country.

Min-Jue XieDivision of Cell Biology and Neuroscience, Department of Morphological and Physiological Sciences, Faculty of Medical Sciences, University of Fukui, Fukui, 910-1193, Japan.
Yasuyuki IshikawaDepartment of Systems Life Engineering, Maebashi Institute of Technology, Gunma, 371-0816, Japan.
Hideshi YagiDivision of Cell Biology and Neuroscience, Department of Morphological and Physiological Sciences, Faculty of Medical Sciences, University of Fukui, Fukui, 910-1193, Japan.
Tokuichi IguchiDivision of Cell Biology and Neuroscience, Department of Morphological and Physiological Sciences, Faculty of Medical Sciences, University of Fukui, Fukui, 910-1193, Japan.
Yuichiro OkaDivision of Cell Biology and Neuroscience, Department of Morphological and Physiological Sciences, Faculty of Medical Sciences, University of Fukui, Fukui, 910-1193, Japan.
Kazuki KurodaDivision of Cell Biology and Neuroscience, Department of Morphological and Physiological Sciences, Faculty of Medical Sciences, University of Fukui, Fukui, 910-1193, Japan.
Keiko IwataDivision of Development of Mental Functions, Research Centre for Child Mental Development, University of Fukui, Fukui, 910-1193, Japan.
Hiroshi KiyonariAnimal Resource Development Unit and Genetic Engineering Team, RIKEN Center for Life Science Technologies, Kobe, 650-0047, Japan.
Shinji MatsudaDepartment of Neurophysiology School of Medicine, Keio University, Tokyo, 160-8582, Japan.
Hideo MatsuzakiDivision of Development of Mental Functions, Research Centre for Child Mental Development, University of Fukui, Fukui, 910-1193, Japan.
Michisuke YuzakiDepartment of Neurophysiology School of Medicine, Keio University, Tokyo, 160-8582, Japan.ORCID 0000-0002-5750-3544
Yugo FukazawaDivision of Brain Structures and Function, Department of Morphological and Physiological Sciences, Faculty of Medical Sciences, University of Fukui, Fukui, 910-1193, Japan.ORCID 0000-0001-7436-8797
Makoto SatoDivision of Cell Biology and Neuroscience, Department of Morphological and Physiological Sciences, Faculty of Medical Sciences, University of Fukui, Fukui, 910-1193, Japan. makosato@anat2.med.osaka-u.ac.jp.ORCID 0000-0002-6805-8351
University of Fukui · JPKeio University · JPMaebashi Institute of Technology · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The essential involvement of phosphoinositides in synaptic plasticity is well-established, but incomplete knowledge of the downstream molecular entities prevents us from understanding their signalling cascades completely. Here, we determined that Phldb2, of which pleckstrin-homology domain is highly sensitive to PIP

Indexed as

Carrier ProteinsDisks Large Homolog 4 ProteinLong-Term PotentiationMembrane ProteinsPhosphatidylinositol PhosphatesReceptors, AMPAReceptors, N-Methyl-D-AspartateAnimalsCalcium-Calmodulin-Dependent Protein Kinase Type 2MemoryMiceMicrofilament ProteinsNeuronal PlasticityProtein BindingCalcium-Calmodulin-Dependent Protein Kinase Type 2Carrier ProteinsDisks Large Homolog 4 ProteinMembrane ProteinsMicrofilament ProteinsPhldb2 protein, mousephosphatidylinositol 3-phosphatePhosphatidylinositol PhosphatesReceptors, AMPAReceptors, N-Methyl-D-Aspartate

Identifiers

PMID30867511
PMCPMC6416313
OpenAlexW2921665589

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.