Evidence map›Paper›PMID 30866049›Full record

ReviewImmunology2019

Immune- and non-immune-mediated roles of regulatory T-cells during wound healing.

Dietmar M Zaiss, Carlos M Minutti, Johanna A Knipper

Open access · bronzeAbstract readReview
In one paragraph

Review in Immunology, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 49 papers.

0numbers the graph read from it
0cells of the map it votes in
49citing papers in PubMed
3.4field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

49 citing papers in PubMed, 73 citations in OpenAlex.

  1. Regulatory T cells in the skin: Origin, function, and plasticity.JID innovations : skin science from molecules to population health · 2026
    Review
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  4. Article
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  6. Review
  7. Article
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  9. Amphiregulin in Fibrotic Diseases and Cancer.International journal of molecular sciences · 2025
    Review
  10. Review
  11. Article
  12. Article
  13. Review
  14. Foxp3Experimental eye research · 2024
    Article
  15. Article
  16. Article
  17. Review
  18. Article
  19. Review
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

Dietmar M ZaissInstitute of Immunology and Infection Research, University of Edinburgh, Edinburgh, UK.ORCID 0000-0003-3596-7062
Carlos M MinuttiInstitute of Immunology and Infection Research, University of Edinburgh, Edinburgh, UK.ORCID 0000-0002-9663-1928
Johanna A KnipperInstitute of Immunology and Infection Research, University of Edinburgh, Edinburgh, UK.ORCID 0000-0002-9038-0566
Centre for Immunity, Infection and Evolution · GBThe Francis Crick Institute · GB

Funding

Medical Research Council MR/M011755/1
6 · The paper itself

Abstract

The immune system has a well-established contribution to tissue homeostasis and wound healing. However, in many cases immune responses themselves can cause severe tissue damage. Thus, the question arose to which extent cells of the immune system directly contribute to the process of wound healing and to which extent the resolution of excessive immune responses may indirectly contribute to wound healing. FoxP3-expressing CD4 T-cells, so-called regulatory T-cells (T

Indexed as

Wound HealingAmphiregulinAnimalsAutocrine CommunicationExtracellular MatrixHumansInterleukin-33Signal TransductionT-Lymphocytes, RegulatoryTransforming Growth Factor betaAmphiregulinAREG protein, humanIL33 protein, humanInterleukin-33Transforming Growth Factor betaAmphiregulincytokines/cytokine receptorsIL-33regulation/suppressionsignal transductiontissue-resident Tregs

Identifiers

PMID30866049
PMCPMC6600083
OpenAlexW2921901296

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.