Evidence map›Paper›PMID 30819085›Full record

ReviewCurrent neuropharmacology2019

Far-infrared Ray-mediated Antioxidant Potentials are Important for Attenuating Psychotoxic Disorders.

Naveen Sharma, Eun-Joo Shin, Nam Hun Kim, Eun-Hee Cho, Bao Trong Nguyen, Ji Hoon Jeong, Choon Gon Jang, Seung-Yeol Nah, Hyoung-Chun Kim

Abstract readReview
In one paragraph

Review in Current neuropharmacology, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed.

  1. Trial
  2. Article
  3. Article
  4. Article
  5. Review
  6. Article
  7. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Naveen SharmaNeuropsychopharmacology and Toxicology Program, BK21 PLUS Project, College of Pharmacy, Kangwon National University, Chunchon 24341, Korea.
Eun-Joo ShinNeuropsychopharmacology and Toxicology Program, BK21 PLUS Project, College of Pharmacy, Kangwon National University, Chunchon 24341, Korea.
Nam Hun KimCollege of Forest and Environmental Sciences, Kangwon National University, Chunchon 24341, Korea.
Eun-Hee ChoDepartment of Internal Medicine, Medical School, Kangwon National University, Chunchon 24341, Korea.
Bao Trong NguyenNeuropsychopharmacology and Toxicology Program, BK21 PLUS Project, College of Pharmacy, Kangwon National University, Chunchon 24341, Korea.
Ji Hoon JeongDepartment of Pharmacology, College of Medicine, Chung-Ang University, Seoul 06974, Korea.
Choon Gon JangDepartment of Pharmacology, School of Pharmacy, Sungkyunkwan University Suwon 16419, Korea.
Seung-Yeol NahGinsentology Research Laboratory and Department of Physiology, College of Veterinary Medicine and Bio/Molecular Informatics Center, Konkuk University, Seoul, Korea.
Hyoung-Chun KimNeuropsychopharmacology and Toxicology Program, BK21 PLUS Project, College of Pharmacy, Kangwon National University, Chunchon 24341, Korea.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Far-infrared ray (FIR) is an electromagnetic wave that produces various health benefits against pathophysiological conditions, such as diabetes mellitus, renocardiovascular disorders, stress, and depression etc. However, the therapeutic application on the FIR-mediated protective potentials remains to be further extended. To achieve better understanding on FIR-mediated therapeutic potentials, we summarized additional findings in the present study that exposure to FIR ameliorates stressful condition, memory impairments, drug dependence, and mitochondrial dysfunction in the central nervous system. In this review, we underlined that FIR requires modulations of janus kinase 2 / signal transducer and activator of transcription 3 (JAK2/STAT3), nuclear factor E2- related factor 2 (Nrf-2), muscarinic M1 acetylcholine receptor (M1 mAChR), dopamine D1 receptor, protein kinase C δ gene, and glutathione peroxidase-1 gene for exerting the protective potentials in response to neuropsychotoxic conditions.

Indexed as

AnimalsAntioxidantsHumansInfrared RaysJanus Kinase 2Nervous System DiseasesNF-E2-Related Factor 2Receptor, Muscarinic M1Receptors, Dopamine D1Signal TransductionAntioxidantsJanus Kinase 2NF-E2-Related Factor 2Receptor, Muscarinic M1Receptors, Dopamine D1dopamine D1 receptorFar-infrared rayglutathione peroxidase-1JAK2/STAT3M1 mAChRneuropsychotoxic conditionsnuclear factor E2-related factor 2protein kinase C δ gene.

Identifiers

PMID30819085
PMCPMC7052827

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.