ReviewFrontiers in human neuroscience2019
Neural Correlates of Interval Timing Deficits in Schizophrenia.
Review in Frontiers in human neuroscience, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.
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Who cites it
9 citing papers in PubMed.
- Effects of divided attention on movement-related cortical potential in community-dwelling elderly adults: A preliminary study.Heliyon · 2024Article
- Not All Mice Are Created Equal: Interval Timing Accuracy and Scalar Timing in 129, Swiss-Webster, and C57Bl/6 Mice.Timing & time perception (Leiden, Netherlands) · 2023Article
- Cilia in the Striatum Mediate Timing-Dependent Functions.Molecular neurobiology · 2023Article
- Core clock gene,Animal cells and systems · 2023Article
- Ramping activity in the striatum.Frontiers in computational neuroscience · 2022Article
- Parallel processing of working memory and temporal information by distinct types of cortical projection neurons.Nature communications · 2021Article
- Movement-Related Potentials Associated with Motor Timing Errors as Determined by Internally Cued Movement Onset.Psychiatry investigation · 2021Article
- Effects of a dual task and different levels of divided attention on motor-related cortical potential.Journal of physical therapy science · 2020Article
- Sustaining temporal attention prevents habit expression during operant learning in rats.Scientific reports · 2020Article
Corrections and comments
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Authors and funding
2 authors.
Funding
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Abstract
Previous research has shown that schizophrenia (SZ) patients exhibit impairments in interval timing. The cause of timing impairments in SZ remains unknown but may be explained by a dysfunction in the fronto-striatal circuits. Although the current literature includes extensive behavioral data on timing impairments, there is limited focus on the neural correlates of timing in SZ. The neuroimaging literature included in the current review reports hypoactivation in the dorsal-lateral prefrontal cortex (DLPFC), supplementary motor area (SMA) and the basal ganglia (BG). Timing deficits and deficits in attention and working memory (WM) in SZ are likely due to a dysfunction of dopamine (DA) and gamma-aminobutyric acid (GABA) neurotransmission in the cortico-striatal-thalamo-cortical circuits, which are highly implicated in executive functioning and motor preparation.
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Registered trials
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