ArticleEnvironmental science and pollution research international2019
Inhalation cancer risk estimation of source-specific personal exposure for particulate matter-bound polycyclic aromatic hydrocarbons based on positive matrix factorization.
Article in Environmental science and pollution research international, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
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Who cites it
4 citing papers in PubMed.
- Optimization of Cancer Risk Assessment Models for PMToxics · 2022Article
- Characteristics, Source Contributions, and Source-Specific Health Risks of PMInternational journal of environmental research and public health · 2022Article
- Assessing Approaches of Human Inhalation Exposure to Polycyclic Aromatic Hydrocarbons: A Review.International journal of environmental research and public health · 2021Review
- PMInternational journal of environmental research and public health · 2020Article
Corrections and comments
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Authors and funding
11 authors.
Funding
Abstract
In previous studies, inhalation cancer risk was estimated using conventional risk assessment method, which was normally based on compound-specific analysis, and cannot provide substantial data for source-specific particulate matter concentrations and pollution control. In the present study, we applied an integrated risk analysis method, which was a synthetic combination of source apportionment receptor model and risk assessment method, to estimate cancer risks associated to individual PAHs coming from specific sources. Personal exposure particulate matter samples referring to an elderly panel were collected in a community of Tianjin, Northern China, in 2009, and 12 PAH compounds were measured using GC-MS. Positive matrix factorization (PMF) was used to extract the potential sources and quantify the source contributions to the PAH mixture. Then, the lung cancer risk of each modeled source was estimated by summing up the cancer risks of all measured PAH species according to the extracted source profile. The final results indicated that the overall cancer risk was 1.12 × 10
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Registered trials
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