Evidence map›Paper›PMID 30744123›Full record

ArticleNutrients2019

Activation of Lipid Mediator Formation Due to Lipoprotein Apheresis.

Karsten-H Weylandt, Christoph Schmöcker, Annika I Ostermann, Laura Kutzner, Ina Willenberg, Stefanie Kiesler, Elisabeth Steinhagen-Thiessen, Nils Helge Schebb, Ursula Kassner

Open access · goldAbstract read
In one paragraph

Article in Nutrients, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.7field-weighted citation impact, top 34% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 7 citations in OpenAlex.

  1. Article
  2. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 4 institutions in 1 country.

Karsten-H WeylandtMedical Department, Divisions of Hepatology, Gastroenterology, Oncology, Hematologyand Diabetes, Ruppiner Kliniken, Brandenburg Medical School, 16816 Neuruppin, Germany. karsten.weylandt@charite.de.
Christoph SchmöckerMedical Department, Divisions of Hepatology, Gastroenterology, Oncology, Hematologyand Diabetes, Ruppiner Kliniken, Brandenburg Medical School, 16816 Neuruppin, Germany. christoph.schmoecker@mhb-fontane.de.
Annika I OstermannInstitute for Food Toxicology and Analytical Chemistry, University of Veterinary Medicine Hannover, 30173 Hannover, Germany. annika.ostermann@schebb-web.de.
Laura KutznerInstitute for Food Toxicology and Analytical Chemistry, University of Veterinary Medicine Hannover, 30173 Hannover, Germany. l.kutzner@gmx.de.
Ina WillenbergInstitute for Food Toxicology and Analytical Chemistry, University of Veterinary Medicine Hannover, 30173 Hannover, Germany. ina.willenberg@gmail.com.
Stefanie KieslerMedical Department, Divisions of Hepatology, Gastroenterology, Oncology, Hematologyand Diabetes, Ruppiner Kliniken, Brandenburg Medical School, 16816 Neuruppin, Germany. S.Kiesler@ruppiner-kliniken.de.
Elisabeth Steinhagen-ThiessenCharité-Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, Medical Department, Division of Hepatology and Gastroenterology (including Metabolic Diseases), Campus Virchow Klinikum, 13353 Berlin, Germany. elisabeth.Steinhagen-Thiessen@charite.de.
Nils Helge SchebbInstitute for Food Toxicology and Analytical Chemistry, University of Veterinary Medicine Hannover, 30173 Hannover, Germany. nils@schebb-web.de.
Ursula KassnerCharité-Universitätsmedizin Berlin, corporate member of Freie Universität Berlin, Humboldt-Universität zu Berlin, Medical Department, Division of Hepatology and Gastroenterology (including Metabolic Diseases), Campus Virchow Klinikum, 13353 Berlin, Germany. ursula.Kassner@charite.de.
Humboldt-Universität zu Berlin · DEUniversity of Veterinary Medicine Hannover, Foundation · DEMedizinische Hochschule Brandenburg Theodor Fontane · DEUniversity of Wuppertal · DE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Lipoprotein apheresis reliably reduces low-density lipoprotein (LDL) cholesterol in patients with atherosclerotic disease and therapy-refractory hypercholesterolemia or elevated lipoprotein (a) (Lp(a)). Besides lowering lipoproteins and triglycerides, apheresis also decreases levels of essential omega-6 and omega-3 polyunsaturated fatty acids (

Indexed as

Blood Component RemovalChromatography, LiquidFatty Acids, Omega-3Fatty Acids, Omega-6FemaleGas Chromatography-Mass SpectrometryHeparinHumansLipoproteins, LDLMaleMiddle AgedTandem Mass SpectrometryFatty Acids, Omega-3Fatty Acids, Omega-6HeparinLipoproteins, LDLdocosahexaenoic acidgas chromatographyhyperlipoproteinaemiaLC-MS/MSlipid apheresislipid mediatorslipidomicsn-3 PUFAn-6 PUFA.omega-3 polyunsaturated fatty acids, eicosapentaenoic acidoxylipins

Identifiers

PMID30744123
PMCPMC6412478
OpenAlexW2911438507

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.