ReviewInternational journal of molecular sciences2019
Abiotic Stresses Intervene with ABA Signaling to Induce Destructive Metabolic Pathways Leading to Death: Premature Leaf Senescence in Plants.
Review in International journal of molecular sciences, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 47 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
47 citing papers in PubMed, 128 citations in OpenAlex.
- Tomato Leaf Color Diversity as a Functional Trait: Molecular Mechanisms, Physiological Significance, and Environmental Responses.International journal of molecular sciences · 2026Review
- From signals to solutions: stress-induced leaf senescence and synthetic biology and AI approaches for crop resilience.Molecular horticulture · 2026Review
- Molecular and Physiological Regulation of Premature Leaf Senescence in Rice.Plants (Basel, Switzerland) · 2026Review
- Application of Beneficial Bacteria to Enhance Plant Drought Resilience.Plants (Basel, Switzerland) · 2026Review
- Integrative physiological and transcriptomic analysis reveals drought response mechanisms in Abrus mollis Hance.BMC plant biology · 2026Article
- Exogenously applied ABA alleviates dysplasia of maize (Plant signaling & behavior · 2025Article
- Pan-Genome-Based Characterization of the PYL Transcription Factor Family inPlants (Basel, Switzerland) · 2025Article
- Natural variation of CTB5 confers cold adaptation in plateau japonica rice.Nature communications · 2025Article
- Ubiquitin LigaseInternational journal of molecular sciences · 2024Article
- Knockdown ofPlants (Basel, Switzerland) · 2024Article
- Screening and Identification of Target Gene ofInternational journal of molecular sciences · 2024Article
- Comparative transcriptome analysis reveals major genes, transcription factors and biosynthetic pathways associated with leaf senescence in rice under different nitrogen application.BMC plant biology · 2024Article
- Apple E3 ligase MdPUB23 mediates ubiquitin-dependent degradation of MdABI5 to delay ABA-triggered leaf senescence.Horticulture research · 2024Article
- KAKU4 regulates leaf senescence through modulation of H3K27me3 deposition in the Arabidopsis genome.BMC plant biology · 2024Article
- Impact of irrigation, nitrogen fertilization, and plant density on stay-green and its effects on agronomic traits in maize.Frontiers in plant science · 2024Article
- Article
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- Calcium in the Life Cycle of Legume Root Nodules.Indian journal of microbiology · 2023Review
- Arabidopsis Protein Phosphatase PIA1 Impairs Plant Drought Tolerance by Serving as a Common Negative Regulator in ABA Signaling Pathway.Plants (Basel, Switzerland) · 2023Article
- Morphological and Physiological Mechanisms of Melatonin on Delaying Drought-Induced Leaf Senescence in Cotton.International journal of molecular sciences · 2023Article
Corrections and comments
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Authors and funding
6 authors at 2 institutions in 1 country.
Funding
Abstract
Abiotic stresses trigger premature leaf senescence by affecting some endogenous factors, which is an important limitation for plant growth and grain yield. Among these endogenous factors that regulate leaf senescence, abscisic acid (ABA) works as a link between the oxidase damage of cellular structure and signal molecules responding to abiotic stress during leaf senescence. Considering the importance of ABA, we collect the latest findings related to ABA biosynthesis, ABA signaling, and its inhibitory effect on chloroplast structure destruction, chlorophyll (Chl) degradation, and photosynthesis reduction. Post-translational changes in leaf senescence end with the exhaustion of nutrients, yellowing of leaves, and death of senescent tissues. In this article, we review the literature on the ABA-inducing leaf senescence mechanism in rice and
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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.