Evidence map›Paper›PMID 30558867›Full record

ArticleNeuroImage. Clinical2019

Differences in hippocampal subfield volume are seen in phenotypic variants of early onset Alzheimer's disease.

Thomas D Parker, Catherine F Slattery, Keir X X Yong, Jennifer M Nicholas, Ross W Paterson, Alexander J M Foulkes, Ian B Malone, David L Thomas, David M Cash, Sebastian J Crutch and 2 more

Open access · goldAbstract read
In one paragraph

Article in NeuroImage. Clinical, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 27 papers.

0numbers the graph read from it
0cells of the map it votes in
27citing papers in PubMed
4.1field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

27 citing papers in PubMed, 53 citations in OpenAlex.

  1. Trial
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  5. Reply to Letter to Editor.Experimental brain research · 2023
    Article
  6. Article
  7. Hippocampal subfield morphology in regular hemodialysis patients.Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association · 2023
    Article
  8. Article
  9. Article
  10. Article
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  12. Volumetric changes within hippocampal subfields in Alzheimer's disease continuum.Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology · 2022
    Article
  13. Review
  14. Article
  15. Review
  16. Article
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  18. Observational
  19. Recent Advances in Imaging of Preclinical, Sporadic, and Autosomal Dominant Alzheimer's Disease.Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics · 2021
    Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 4 institutions in 2 countries.

Thomas D ParkerDepartment of Neurodegenerative Disease, Queen Square Institute of Neurology, UCL, London, UK. Electronic address: thomas.parker@ucl.ac.uk.
Catherine F SlatteryDepartment of Neurodegenerative Disease, Queen Square Institute of Neurology, UCL, London, UK.
Keir X X YongDepartment of Neurodegenerative Disease, Queen Square Institute of Neurology, UCL, London, UK.
Jennifer M NicholasDepartment of Neurodegenerative Disease, Queen Square Institute of Neurology, UCL, London, UK; Department of Medical Statistics, London School of Hygiene and Tropical Medicine, London, UK.
Ross W PatersonDepartment of Neurodegenerative Disease, Queen Square Institute of Neurology, UCL, London, UK.
Alexander J M FoulkesDepartment of Neurodegenerative Disease, Queen Square Institute of Neurology, UCL, London, UK.
Ian B MaloneDepartment of Neurodegenerative Disease, Queen Square Institute of Neurology, UCL, London, UK.
David L ThomasNeuroradiological Academic Unit, Department of Brain Repair and Rehabilitation, UCL Institute of Neurology, London, UK; Leonard Wolfson Experimental Neurology Centre, UCL Institute of Neurology, London, UK.
David M CashDepartment of Neurodegenerative Disease, Queen Square Institute of Neurology, UCL, London, UK.
Sebastian J CrutchDepartment of Neurodegenerative Disease, Queen Square Institute of Neurology, UCL, London, UK.
Nick C FoxDepartment of Neurodegenerative Disease, Queen Square Institute of Neurology, UCL, London, UK.
Jonathan M SchottDepartment of Neurodegenerative Disease, Queen Square Institute of Neurology, UCL, London, UK.
National Hospital for Neurology and Neurosurgery · GBInstitute of Ophthalmology · MXMRC Prion Unit · GBUniversity of London · GB

Funding

Medical Research Council MR/M009106/1Medical Research Council MR/M023664/1Wellcome Trust
6 · The paper itself

Abstract

The most common presentation of early onset Alzheimer's disease (EOAD - defined as symptom onset <65 years) is with progressive episodic memory impairment - amnestic or typical Alzheimer's disease (tAD). However, EOAD is notable for its phenotypic heterogeneity, with posterior cortical atrophy (PCA) - characterised by prominent higher-order visual processing deficits and relative sparing of episodic memory - the second most common canonical phenotype. The hippocampus, which comprises a number of interconnected anatomically and functionally distinct subfields, is centrally involved in Alzheimer's disease and is a crucial mediator of episodic memory. The extent to which volumes of individual hippocampal subfields differ between different phenotypes in EOAD is unclear. The aim of this analysis was to investigate the hypothesis that patients with a PCA phenotype will exhibit differences in specific hippocampal subfield volumes compared to tAD. We studied 63 participants with volumetric T1-weighted MRI performed on the same 3T scanner: 39 EOAD patients [27 with tAD and 12 with PCA] and 24 age-matched controls. Volumetric estimates of the following hippocampal subfields for each participant were obtained using Freesurfer version 6.0: CA1, CA2/3, CA4, presubiculum, subiculum, hippocampal tail, parasubiculum, the molecular and granule cell layers of the dentate gryus (GCMLDG), the molecular layer, and the hippocampal amygdala transition area (HATA). Linear regression analyses comparing mean hippocampal subfield volumes between groups, adjusting for age, sex and head size, were performed. Using a Bonferonni-corrected p-value of p < 0.0025, compared to controls, tAD was associated with atrophy in all hippocampal regions, except the parasubiculum. In PCA patients compared to controls, the strongest evidence for volume loss was in the left presubiclum, right subiculum, right GCMLDG, right molecular layer and the right HATA. Compared to PCA, patients with tAD had strong evidence for smaller volumes in left CA1 and left hippocampal tail. In conclusion, these data provide evidence that hippocampal subfield volumes differ in different phenotypes of EOAD.

Indexed as

PhenotypeAgedAlzheimer DiseaseFemaleGenetic VariationHippocampusHumansMaleMiddle AgedOrgan SizeAtypical Alzheimer's diseaseCA1Early-onset Alzheimer's diseaseHippocampal subfieldsPosterior cortical atrophyPresubiculum

Identifiers

PMID30558867
PMCPMC6411912
OpenAlexW2905420408

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.