ArticlePlant signaling & behavior2019
Contribution of KUPs to potassium and cesium accumulation appears complementary in Arabidopsis.
Article in Plant signaling & behavior, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
7 citing papers in PubMed, 13 citations in OpenAlex.
- Suppressing radiocesium uptake and root-to-shoot translocation in crops: Molecular, agronomic, and ecological strategies.Environmental geochemistry and health · 2026Review
- Arabidopsis transcriptomic analysis reveals cesium inhibition of root growth involves abscisic acid signaling.Planta · 2024Article
- Potassium transporter KUP9 participates in KStress biology · 2022Article
- Genome-wide characterization and expression analysis of HAK K3 Biotech · 2021Article
- Syringic Acid Alleviates Cesium-Induced Growth Defect inInternational journal of molecular sciences · 2020Article
- Article
- Why Nature Chose Potassium.Journal of molecular evolution · 2019Review
Corrections and comments
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Authors and funding
3 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Cesium has no known beneficial effects on plants and while plants have the ability to absorb it through the root system, plant growth is retarded at high concentrations. Recently, we have shown that potassium influx through a potassium channel complex AKT1-KC1 is inhibited by cesium in Arabidopsis thaliana and the resultant reduction in potassium accumulation in the plant is the primary cause of retarded growth. By contrast, a major potassium transporter, HAK5 whose function is crucial under potassium deficiency, was found to be either not affected or complementary under cesium stress in the low affinity potassium range. Here we show the effects of insertional mutation on other members of KUP/HAK/KT gene family in response to cesium stress. Potassium and cesium concentrations in each mutant line demonstrated that disruption of a single KUP/HAK/KT gene was not sufficient to significantly reduce potassium/cesium accumulation, suggesting a complementary effect among these KUP (K
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Registered trials
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