Evidence map›Paper›PMID 30505654›Full record

ReviewActa pharmaceutica Sinica. B2018

Yi Niu, Arabella Wan, Ziyou Lin, Xiongbin Lu, Guohui Wan

Open access · diamondAbstract readReview
In one paragraph

Review in Acta pharmaceutica Sinica. B, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 46 papers.

0numbers the graph read from it
0cells of the map it votes in
46citing papers in PubMed
4.5field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

46 citing papers in PubMed, 95 citations in OpenAlex.

  1. Review
  2. NBMC medical genomics · 2025
    Article
  3. Overexpression ofOncology letters · 2025
    Article
  4. Article
  5. Review
  6. Insights into the role of RNA mGenes & diseases · 2024
    Review
  7. Non-coding RNA-Mediated N6-Methyladenosine (mNon-coding RNA research · 2024
    Review
  8. Article
  9. Targeted mJournal of advanced research · 2024
    Article
  10. The role of mCell death discovery · 2023
    Article
  11. Roles and implications of mRNA NCancer communications (London, England) · 2023
    Review
  12. N6-methyladenosine demethylase FTO enhances chemo-resistance in colorectal cancer through SIVA1-mediated apoptosis.Molecular therapy : the journal of the American Society of Gene Therapy · 2023
    Article
  13. Research Progress of mBioMed research international · 2023
    Review
  14. mEpigenetics · 2022
    Article
  15. Article
  16. Impact of N6-methyladenosine (mCell communication and signaling : CCS · 2022
    Review
  17. The mActa pharmacologica Sinica · 2022
    Article
  18. Biological roles of RNA mBiomarker research · 2022
    Review
  19. Review
  20. Modifying the mMolecular psychiatry · 2021
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 3 institutions in 2 countries.

Yi NiuSchool of Pharmaceutical Sciences, Sun Yat-Sen University, Guangzhou 510006, China.
Arabella WanSchool of Medical Laboratory Science, Pennsylvania Hospital, Philadelphia, PA 19107, USA.
Ziyou LinSchool of Pharmaceutical Sciences, Sun Yat-Sen University, Guangzhou 510006, China.
Xiongbin LuDepartment of Medical and Molecular Genetics, Indiana University School of Medicine, Indianapolis, IN 46202, USA.
Guohui WanSchool of Pharmaceutical Sciences, Sun Yat-Sen University, Guangzhou 510006, China.
Sun Yat-sen University · CNIndiana University Health · USPennsylvania Hospital · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

PubMed holds no abstract for this paper.

Indexed as

Drug developmentHuman cancerm6A modification regulatorN6-MethyladenosinePharmacological target

Identifiers

PMID30505654
PMCPMC6251950
OpenAlexW2807347076

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.