Evidence map›Paper›PMID 30487302›Full record

ReviewOpen biology2018

Cellular plasticity, caspases and autophagy; that which does not kill us, well, makes us different.

Tin Tin Su

Abstract readReview
In one paragraph

Review in Open biology, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Article
  2. A not-so-simple twist of fate.Developmental cell · 2021
    Article
  3. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

Tin Tin SuDepartment of Molecular, Cellular and Developmental Biology, 347 UCB, University of Colorado, Boulder, CO 80309-0347, USA tin.su@colorado.edu.ORCID 0000-0003-0139-4390

Funding

Cellular Plasticity and Regeneration after Radiation Damage in DrosophilaR35GM130374 · NIGMS · UNIVERSITY OF COLORADO · PI Tin Tin Su · 2019 to 2026
$3.2M
Consequences of Cell Death in DrosophilaR01GM106317 · NIGMS · UNIVERSITY OF COLORADO · PI SU, TIN TIN · 2014 to 2017
$1.2M
NIGMS NIH HHS R01 GM106317NIGMS NIH HHS R35 GM130374
6 · The paper itself

Abstract

The ability to regenerate is a fundamental requirement for tissue homeostasis. Regeneration draws on three sources of cells. First and best-studied are dedicated stem/progenitor cells. Second, existing cells may proliferate to compensate for the lost cells of the same type. Third, a different cell type may change fate to compensate for the lost cells. This review focuses on regeneration of the third type and will discuss the contributions by post-transcriptional mechanisms including the emerging evidence for cell-autonomous and non-lethal roles of cell death pathways.

Indexed as

AnimalsAutophagyCaspasesHumansRegenerationStem CellsCaspasesautophagycaspasemicroRNAtransdifferentiation

Identifiers

PMID30487302
PMCPMC6282069

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.