Evidence map›Paper›PMID 30405617›Full record

ArticleFrontiers in immunology2018

Human Monocytic Suppressive Cells Promote Replication of

Neha Agrawal, Ioana Streata, Gang Pei, January Weiner, Leigh Kotze, Silke Bandermann, Laura Lozza, Gerhard Walzl, Nelita du Plessis, Mihai Ioana and 2 more

Open access · goldAbstract read
In one paragraph

Article in Frontiers in immunology, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 26 papers.

0numbers the graph read from it
0cells of the map it votes in
26citing papers in PubMed
2.0field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

26 citing papers in PubMed, 31 citations in OpenAlex.

  1. Review
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  6. Review
  7. Suppressive myeloid cells in SARS-CoV-2 andFrontiers in immunology · 2023
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 5 institutions in 3 countries.

Neha AgrawalMax Planck Institute for Infection Biology, Department of Immunology, Berlin, Germany.
Ioana StreataUniversity of Medicine and Pharmacy Craiova, Human Genomics Laboratory, Craiova, Romania.
Gang PeiMax Planck Institute for Infection Biology, Department of Immunology, Berlin, Germany.
January WeinerMax Planck Institute for Infection Biology, Department of Immunology, Berlin, Germany.
Leigh KotzeDivision of Molecular Biology and Human Genetics, Department of Biomedical Sciences, Faculty of Medicine and Health Sciences, SAMRC Centre for Tuberculosis Research, DST and NRF Centre of Excellence for Biomedical TB Research, Stellenbosch University, Tygerberg, South Africa.
Silke BandermannMax Planck Institute for Infection Biology, Department of Immunology, Berlin, Germany.
Laura LozzaMax Planck Institute for Infection Biology, Department of Immunology, Berlin, Germany.
Gerhard WalzlDivision of Molecular Biology and Human Genetics, Department of Biomedical Sciences, Faculty of Medicine and Health Sciences, SAMRC Centre for Tuberculosis Research, DST and NRF Centre of Excellence for Biomedical TB Research, Stellenbosch University, Tygerberg, South Africa.
Nelita du PlessisDivision of Molecular Biology and Human Genetics, Department of Biomedical Sciences, Faculty of Medicine and Health Sciences, SAMRC Centre for Tuberculosis Research, DST and NRF Centre of Excellence for Biomedical TB Research, Stellenbosch University, Tygerberg, South Africa.
Mihai IoanaUniversity of Medicine and Pharmacy Craiova, Human Genomics Laboratory, Craiova, Romania.
Stefan H E KaufmannMax Planck Institute for Infection Biology, Department of Immunology, Berlin, Germany.
Anca DorhoiMax Planck Institute for Infection Biology, Department of Immunology, Berlin, Germany.
Max Planck Institute for Infection Biology · DEStellenbosch University · ZAUniversity of Medicine and Pharmacy of Craiova · ROSouth African Medical Research Council · ZAUniversität Greifswald · DE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Tuberculosis (TB) has tremendous public health relevance. It most frequently affects the lung and is characterized by the development of unique tissue lesions, termed granulomas. These lesions encompass various immune populations, with macrophages being most extensively investigated. Myeloid derived suppressor cells (MDSCs) have been recently identified in TB patients, both in the circulation and at the site of infection, however their interactions with

Indexed as

B7-H1 AntigenBacterial LoadCell ProliferationCells, CulturedDNA ReplicationExtracellular Signal-Regulated MAP KinasesGranuloma, Respiratory TractHumansImmune ToleranceInterleukin-10LymphocytesMycobacterium tuberculosisMyeloid-Derived Suppressor CellsNF-kappa BSignal TransductionTuberculosis, PulmonaryB7-H1 AntigenExtracellular Signal-Regulated MAP KinasesInterleukin-10NF-kappa BgranulomaIL-10Mycobacterium tuberculosismyeloid-derived suppressor cellsPD-L1tuberculosis

Identifiers

PMID30405617
PMCPMC6205994
OpenAlexW2897233210

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.