Evidence map›Paper›PMID 30373150›Full record

ReviewInternational journal of molecular sciences2018

Emi Nagoshi

Open access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 27 papers.

0numbers the graph read from it
0cells of the map it votes in
27citing papers in PubMed
4.6field-weighted citation impact, top 5% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

27 citing papers in PubMed, 61 citations in OpenAlex.

  1. Article
  2. Article
  3. Royal Society open science · 2025
    Article
  4. Review
  5. Article
  6. Review
  7. Article
  8. Article
  9. Review
  10. Article
  11. A new metabolic model ofLife science alliance · 2023
    Article
  12. Asymmetric Presynaptic Depletion of Dopamine Neurons in aInternational journal of molecular sciences · 2023
    Article
  13. Journal of biosciences · 2023
    Review
  14. International journal of molecular sciences · 2022
    Article
  15. Article
  16. Article
  17. Article
  18. Article
  19. Article
  20. The Importance ofCells · 2021
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author at 1 institution in 1 country.

Emi NagoshiDepartment of Genetics and Evolution, Sciences III, University of Geneva, 30 Quai Ernest-Ansermet, CH-1211 Geneva-4, Switzerland. Emi.Nagoshi@unige.ch.ORCID 0000-0002-6365-4074
University of Geneva · CH

Funding

European Research Council ERC-2012-StG_20111109_311194Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung 31003A_169548
6 · The paper itself

Abstract

Parkinson's disease (PD) is the most common cause of movement disorders and is characterized by the progressive loss of dopaminergic neurons in the substantia nigra. It is increasingly recognized as a complex group of disorders presenting widely heterogeneous symptoms and pathology. With the exception of the rare monogenic forms, the majority of PD cases result from an interaction between multiple genetic and environmental risk factors. The search for these risk factors and the development of preclinical animal models are in progress, aiming to provide mechanistic insights into the pathogenesis of PD. This review summarizes the studies that capitalize on modeling sporadic (i.e., nonfamilial) PD using

Indexed as

Disease Models, AnimalDrosophila melanogasterAnimalsGenetic Predisposition to DiseaseHumansParkinson DiseaseParkinson Disease, Secondaryanimal modelDrosophilasporadic Parkinson’s disease

Identifiers

PMID30373150
PMCPMC6275057
OpenAlexW2890276391

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.