Evidence map›Paper›PMID 30345294›Full record

ReviewBioMed research international2018

Origin of the Autophagosome Membrane in Mammals.

Yun Wei, Meixia Liu, Xianxiao Li, Jiangang Liu, Hao Li

Abstract readReview
In one paragraph

Review in BioMed research international, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 50 papers.

0numbers the graph read from it
0cells of the map it votes in
50citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

50 citing papers in PubMed.

  1. Article
  2. Review
  3. Article
  4. Article
  5. Review
  6. Article
  7. Review
  8. Review
  9. Article
  10. Review
  11. Article
  12. Article
  13. Interaction between coronaviruses and the autophagic response.Frontiers in cellular and infection microbiology · 2024
    Review
  14. Article
  15. The Role of Alternative Mitophagy in Heart Disease.International journal of molecular sciences · 2023
    Review
  16. Review
  17. Programmed Cell Death in Diabetic Nephropathy: A Review of Apoptosis, Autophagy, and Necroptosis.Medical science monitor : international medical journal of experimental and clinical research · 2022
    Review
  18. Article
  19. Review
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Yun WeiXiyuan Hospital of China Academy of Chinese Medical Sciences, Beijing 100091, China.ORCID 0000-0001-9270-2701
Meixia LiuXiyuan Hospital of China Academy of Chinese Medical Sciences, Beijing 100091, China.ORCID 0000-0003-3277-8004
Xianxiao LiDepartment of Oncology, Air Force General Hospital, Beijing 100142, China.
Jiangang LiuXiyuan Hospital of China Academy of Chinese Medical Sciences, Beijing 100091, China.ORCID 0000-0002-2313-4592
Hao LiXiyuan Hospital of China Academy of Chinese Medical Sciences, Beijing 100091, China.ORCID 0000-0001-8502-2556

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Autophagy begins with the nucleation of phagophores, which then expand to give rise to the double-membrane autophagosomes. Autophagosomes ultimately fuse with lysosomes, where the cytosolic cargoes are degraded. Accumulation of autophagosomes is a hallmark of autophagy and neurodegenerative disorders including Alzheimer's and Huntington's disease. In recent years, the sources of autophagosome membrane have attracted a great deal of interests, even so, the membrane donors for autophagosomes are still under debate. In this review, we describe the probable sources of autophagosome membrane.

Indexed as

Alzheimer DiseaseAnimalsAutophagosomesAutophagyHumansHuntington DiseaseLysosomes

Identifiers

PMID30345294
PMCPMC6174804

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.