Evidence map›Paper›PMID 30340432›Full record

ReviewInternational journal of molecular sciences2018

Gas Signaling Molecules and Mitochondrial Potassium Channels.

Agnieszka Walewska, Adam Szewczyk, Piotr Koprowski

Open access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 19 papers.

0numbers the graph read from it
0cells of the map it votes in
19citing papers in PubMed
2.2field-weighted citation impact, top 14% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

19 citing papers in PubMed, 46 citations in OpenAlex.

  1. Article
  2. Review
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  4. Plants and Mushrooms as Possible New Sources of HInternational journal of molecular sciences · 2023
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Agnieszka WalewskaLaboratory of Intracellular Ion Channels, Nencki Institute of Experimental Biology PAS, 3 Pasteur St., 02-093 Warsaw, Poland. a.walewska@nencki.gov.pl.
Adam SzewczykLaboratory of Intracellular Ion Channels, Nencki Institute of Experimental Biology PAS, 3 Pasteur St., 02-093 Warsaw, Poland. a.szewczyk@nencki.gov.pl.ORCID 0000-0001-5519-260X
Piotr KoprowskiLaboratory of Intracellular Ion Channels, Nencki Institute of Experimental Biology PAS, 3 Pasteur St., 02-093 Warsaw, Poland. p.koprowski@nencki.gov.pl.
Instytut Biologii Doświadczalnej im. Marcelego Nenckiego · PL

Funding

Narodowe Centrum Nauki 2015/17/B/NZ1/02496, 2015/19/B/NZ1/02794
6 · The paper itself

Abstract

Recently, gaseous signaling molecules, such as carbon monoxide (CO), nitric oxide (NO), and hydrogen sulfide (H₂S), which were previously considered to be highly toxic, have been of increasing interest due to their beneficial effects at low concentrations. These so-called gasotransmitters affect many cellular processes, such as apoptosis, proliferation, cytoprotection, oxygen sensing, ATP synthesis, and cellular respiration. It is thought that mitochondria, specifically their respiratory complexes, constitute an important target for these gases. On the other hand, increasing evidence of a cytoprotective role for mitochondrial potassium channels provides motivation for the analysis of the role of gasotransmitters in the regulation of channel function. A number of potassium channels have been shown to exhibit activity within the inner mitochondrial membrane, including ATP-sensitive potassium channels, Ca

Indexed as

Signal TransductionAnimalsBiomarkersCarbon MonoxideGasesHumansHydrogen SulfideMitochondriaNitric OxidePotassium ChannelsProtein BindingBiomarkersCarbon MonoxideGasesHydrogen SulfideNitric OxidePotassium ChannelsBKCa channelcarbon monoxidegasotransmittershemehydrogen sulfideKATP channelmitochondrianitric oxidepotassium channels

Identifiers

PMID30340432
PMCPMC6214077
OpenAlexW2897974085

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.