Evidence map›Paper›PMID 30274168›Full record

ArticleInternational journal of molecular sciences2018

Ectopic Overexpression of Porcine Myh1 Increased in Slow Muscle Fibers and Enhanced Endurance Exercise in Transgenic Mice.

Jin Seop Ahn, Dong-Hwan Kim, Hee-Bok Park, Sang-Hyun Han, Seongsoo Hwang, In-Cheol Cho, Jeong-Woong Lee

Abstract read
In one paragraph

Article in International journal of molecular sciences, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed.

  1. Article
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  9. Conditionally immortalised equine skeletal muscle cell lines forBiochemistry and biophysics reports · 2023
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  13. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Jin Seop AhnBiotherapeutics Translational Research Center, Korea Research Institute of Bioscience and Biotechnology, Daejeon 34141, Korea. ajsws@naver.com.
Dong-Hwan KimBiotherapeutics Translational Research Center, Korea Research Institute of Bioscience and Biotechnology, Daejeon 34141, Korea. dhkim00@kribb.re.kr.
Hee-Bok ParkSubtropical Livestock Research Institute, National Institute of Animal Science, Jeju 63242, Korea. heebokp@korea.kr.
Sang-Hyun HanEducational Science Research Institute, Jeju National University, Jeju 63243, Korea. hansh04@naver.com.ORCID 0000-0002-6615-2811
Seongsoo HwangAnimal Biotechnology Division, National Institute of Animal Science, Wanju 55365, Korea. hwangss@korea.kr.ORCID 0000-0002-0512-7049
In-Cheol ChoAnimal Genetics and Bioinformatics Division, National Institute of Animal Science, Wanju 55365, Korea. choic4753@korea.kr.
Jeong-Woong LeeBiotherapeutics Translational Research Center, Korea Research Institute of Bioscience and Biotechnology, Daejeon 34141, Korea. jwlee@kribb.re.kr.ORCID 0000-0002-2649-9677

Funding

Korea Research Institute of Bioscience and Biotechnology Research Initiative Program
6 · The paper itself

Abstract

Myosin heavy chain (MyHC) isoforms consist of Myh7, Myh2, Myh1, and Myh4, which are expressed in skeletal muscle tissues during postnatal development. These genes influence the contraction⁻relaxation activity in skeletal muscles and are involved in determining muscle composition such as the proportion of fast-to-slow and/or slow-to-fast fiber types. Among them, Myh1 is associated with skeletal muscle contraction and is involved in both slow-to-fast and fast-to-slow transition. However, the muscle transition mechanism is not well understood. For this study, we first produced porcine

Indexed as

Physical Conditioning, AnimalPhysical EnduranceAnimalsGene Expression RegulationMice, Inbred C57BLMice, TransgenicMuscle Fibers, SkeletalMyosin Heavy ChainsSwineMyosin Heavy ChainsMyh1quadricepsslow muscletransgenic mice

Identifiers

PMID30274168
PMCPMC6213911

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.