Evidence map›Paper›PMID 30266744›Full record

ReviewBioscience reports2018

Regulatory effects of lncRNAs and miRNAs on autophagy in malignant tumorigenesis.

Qingqing Yin, Wei Feng, Xianjuan Shen, Shaoqing Ju

Open access · goldAbstract readReview
In one paragraph

Review in Bioscience reports, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed
1.2field-weighted citation impact, top 25% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed, 28 citations in OpenAlex.

  1. Review
  2. Regulation of autophagy by non-coding RNAs in human glioblastoma.Medical oncology (Northwood, London, England) · 2024
    Review
  3. Review
  4. Role of autophagy and its regulation by noncoding RNAs in ovarian cancer.Experimental biology and medicine (Maywood, N.J.) · 2023
    Review
  5. The dual role of autophagy in acute myeloid leukemia.Journal of hematology & oncology · 2022
    Review
  6. Review
  7. Article
  8. Cell death pathways and viruses: Role of microRNAs.Molecular therapy. Nucleic acids · 2021
    Review
  9. Article
  10. Review
  11. Article
  12. Article
  13. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

Qingqing YinCenter of Laboratory Medicine, Affiliated Hospital of Nantong University, Nantong, China.
Wei FengCenter of Laboratory Medicine, Affiliated Hospital of Nantong University, Nantong, China.
Xianjuan ShenSurgical Comprehensive Laboratory, Affiliated Hospital of Nantong University, Nantong, China.
Shaoqing JuCenter of Laboratory Medicine, Affiliated Hospital of Nantong University, Nantong, China jsq814@hotmail.com.ORCID 0000-0001-8192-2274
Nantong University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Autophagy is an important process in endogenous substrate degradation by lysosomes within cells, with a degree of evolutionary conservation. Like apoptosis and cell senescence, cell autophagy is a very important biological phenomenon involving the development and growth of biological processes. Abnormal autophagy may lead to tumorigenesis. In recent years, increasing studies have demonstrated that long non-coding RNAs (lncRNAs) and miRNAs can regulate cell autophagy by modulating targetting gene expression. In this review, we will provide an overview of lncRNAs and miRNAs in autophagy modulation and new insights into the underlying mechanisms, as well as their potential utilization in disease diagnosis, prognosis, and therapy.

Indexed as

AutophagyCarcinogenesisGene Expression Regulation, NeoplasticHumansMicroRNAsNeoplasmsPrognosisRNA, Long NoncodingMicroRNAsRNA, Long Noncodingautophagygene therapylarge intervening non-coding RNAmicroRNAoncogenesis

Identifiers

PMID30266744
PMCPMC6200703
OpenAlexW2894323444

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.