ArticleFood science and biotechnology2016
Kombucha tea prevents obese mice from developing hepatic steatosis and liver damage.
Article in Food science and biotechnology, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.
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Who cites it
10 citing papers in PubMed, 39 citations in OpenAlex.
- Review
- Protective role of hydroalcoholic extract ofResearch in pharmaceutical sciences · 2026Article
- Time and temperature influence on physicochemical, microbiological, and sensory profiles of yerba mate kombucha.Journal of food science and technology · 2024Article
- Hepatoprotective Effect ofJournal of microbiology and biotechnology · 2024Article
- Camellia sinensis methanolic leaves extract: Phytochemical analysis and anticancer activity against human liver cancer cells.PloS one · 2024Article
- Changes in the Bio-Compounds and Biological Activities of Eight Whole Grains Fermentation Starter with Different Oxidized Chin-Shin Oolong Teas.Foods (Basel, Switzerland) · 2023Article
- Kombucha - An ancient fermented beverage with desired bioactivities: A narrowed review.Food chemistry: X · 2022Article
- Hepatoprotective Effect of Kombucha Tea in Rodent Model of Nonalcoholic Fatty Liver Disease/Nonalcoholic Steatohepatitis.International journal of molecular sciences · 2019Article
- Effect of Kombucha on gut-microbiota in mouse having non-alcoholic fatty liver disease.Food science and biotechnology · 2019Article
- Kombucha tea in cancer care: A review of its anti-cancer potential and role in integrative oncology.Avicenna journal of phytomedicineReview
Corrections and comments
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Authors and funding
8 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Nonalcoholic fatty liver disease (NAFLD) is associated with the increased accumulation of hepatocellular lipids. Although Kombucha tea (KT) has emerged as a substance protecting the liver from damage, the effects of KT in NAFLD remain unclear. Hence, we investigated whether KT influenced hepatic steatosis. Db/db mice were fed either control or methionine/choline-deficient (MCD) diets for 4 weeks. The MCD diet group was treated with KT or water for 3 weeks. KT treatment alleviated macrovesicular steatosis compared to the MCD-fed group. The levels of triglyceride, ALT, and AST also decreased in the KT+MCD-treated db/db mice. RNA expression in the MCD+KT group showed reduced triglyceride synthesis and uptake of fatty acids. Immunostaining and western blot assays for active caspase-3 demonstrated a lower level of apoptosis in the MCD+KT than in the MCD group. These results demonstrate that KT attenuated lipid accumulation and protected the liver from damage, promoting liver restoration in mice.
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