Evidence map›Paper›PMID 30237972›Full record

ArticleRespiratory medicine case reports2018

Acute pulmonary embolism and deep vein thrombosis secondary to idiopathic hypereosinophilic syndrome.

Dezhi Li, Li Xu, Dianjie Lin, Shujuan Jiang, Saran Feng, Ling Zhu

Open access · goldAbstract readCase Reports
In one paragraph

Article in Respiratory medicine case reports, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
1.6field-weighted citation impact, top 17% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed, 14 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Hyepereosiniphilic syndrome and COVID-19: 2 case reports.Journal of cardiothoracic surgery · 2023
    Article
  5. Article
  6. Article
  7. Article
  8. Article
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  11. Article
  12. Utility of Blood Cellular Indices in the Risk Stratification of Patients Presenting with Acute Pulmonary Embolism.Clinical and applied thrombosis/hemostasis : official journal of the International Academy of Clinical and Applied Thrombosis/Hemostasis
    Observational
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 3 institutions in 1 country.

Dezhi LiDepartment of Respiratory Medicine, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, China.
Li XuDepartment of Respiratory Medicine, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, China.
Dianjie LinDepartment of Respiratory Medicine, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, China.
Shujuan JiangDepartment of Respiratory Medicine, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, China.
Saran FengDepartment of Hematology, Shandong Provincial Qianfoshan Hospital, Jinan, China.
Ling ZhuDepartment of Respiratory Medicine, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, China.
Shandong Provincial Hospital · CNShandong Provincial QianFoShan Hospital · CNShandong University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Acute pulmonary embolism (PE) is a most dangerous complication that needs prompt treatment to reduce potentially death. There are many well-known prognostic factors indicate the morbidity and mortality in various thromboembolic events. Persistent eosinophilia in peripheral blood can lead to tissue infiltration and even organ damage, but the urgent event of thromboembolism in pulmonary provoked by eosinophil eosinophilia in idiopathic hypereosinophilic syndrome (HES) is relative an unusual presentation. In this paper, we present two cases of patients with multiple PE and deep vein thrombosis secondary to the idiopathic HES. Patients were all treated using anticoagulant therapy and corticosteroids successfully. Accordingly, eosinophilia is another risk and precipitating factor of pulmonary thromboembolism. It is necessary for physicians to make a diagnosis in hypereosinophilia as soon as possible for proper prognosis and in case of further thromboembolic events and prevent end-organ damage.

Indexed as

EmbolismIdiopathic hypereosinophilic syndromePulmonary embolismRisk factorVeins

Identifiers

PMID30237972
PMCPMC6143698
OpenAlexW2891789988

What OpenQuestion holds

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LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.