Evidence map›Paper›PMID 30210182›Full record

ArticleEcological modelling2018

The intrinsic vulnerability of networks to epidemics.

G Strona, C J Carstens, P S A Beck, B A Han

Abstract read
In one paragraph

Article in Ecological modelling, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed.

  1. Article
  2. Review
  3. Article
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

G StronaEuropean Commission, Joint Research Centre, Directorate D - Sustainable Resources - Bio-Economy Unit, 21027 Ispra, VA, Italy.
C J CarstensUniversity of Amsterdam, Korteweg de Vries Instituut, 1098 XG Amsterdam, The Netherlands.
P S A BeckEuropean Commission, Joint Research Centre, Directorate D - Sustainable Resources - Bio-Economy Unit, 21027 Ispra, VA, Italy.
B A HanCary Institute of Ecosystem Studies, Millbrook, NY 12545, USA.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Contact networks are convenient models to investigate epidemics, with nodes and links representing potential hosts and infection pathways, respectively. The outcomes of outbreak simulations on networks are driven both by the underlying epidemic model, and by the networks' structural properties, so that the same pathogen can generate different epidemic dynamics on different networks. Here we ask whether there are general properties that make a contact network intrinsically vulnerable to epidemics (that is, regardless of specific epidemiological parameters). By conducting simulations on a large set of modelled networks, we show that, when a broad range of network topologies is taken into account, the effect of specific network properties on outbreak magnitude is stronger than that of fundamental pathogen features such as transmission rate, infection duration, and immunization ability. Then, by focusing on a large set of real world networks of the same type (potential contacts between field voles,

Indexed as

Contact networksDisease ecologyEmerging infectious diseasesEpidemic modelImmunization

Identifiers

PMID30210182
PMCPMC6039859

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.