Evidence map›Paper›PMID 30197885›Full record

ArticleFrontiers in molecular biosciences2018

Activation of the Calcium-Sensing Receptor Corrects the Impaired Mitochondrial Energy Status Observed in Renal Polycystin-1 Knockdown Cells Modeling Autosomal Dominant Polycystic Kidney Disease.

Annarita Di Mise, Marianna Ranieri, Mariangela Centrone, Maria Venneri, Grazia Tamma, Daniela Valenti, Giovanna Valenti

Open access · goldAbstract read
In one paragraph

Article in Frontiers in molecular biosciences, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed
3.1field-weighted citation impact, top 8% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed, 23 citations in OpenAlex.

  1. The Role of Mitochondria in Polycystic Kidney Disease.International journal of molecular sciences · 2026
    Review
  2. Article
  3. Article
  4. Review
  5. Review
  6. Pre-clinical evaluation of dual targeting of the GPCRs CaSR and V2R as therapeutic strategy for autosomal dominant polycystic kidney disease.FASEB journal : official publication of the Federation of American Societies for Experimental Biology · 2021
    Article
  7. Article
  8. Article
  9. Review
  10. Article
  11. Renal CaInternational journal of molecular sciences · 2019
    Review
  12. Article
  13. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 3 institutions in 1 country.

Annarita Di MiseDepartment of Biosciences, Biotechnologies and Biopharmaceutics, University of Bari, Bari, Italy.
Marianna RanieriDepartment of Biosciences, Biotechnologies and Biopharmaceutics, University of Bari, Bari, Italy.
Mariangela CentroneDepartment of Biosciences, Biotechnologies and Biopharmaceutics, University of Bari, Bari, Italy.
Maria VenneriDepartment of Biosciences, Biotechnologies and Biopharmaceutics, University of Bari, Bari, Italy.
Grazia TammaDepartment of Biosciences, Biotechnologies and Biopharmaceutics, University of Bari, Bari, Italy.
Daniela ValentiInstitute of Biomembranes Bioenergetics and Molecular Biotechnologies, National Council of Research, Bari, Italy.
Giovanna ValentiDepartment of Biosciences, Biotechnologies and Biopharmaceutics, University of Bari, Bari, Italy.
University of Bari Aldo Moro · ITIstituto Nazionale Biostrutture e Biosistemi · ITInstitute of Biomembranes, Bioenergetics and Molecular Biotechnologies · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Autosomal Dominant Polycistic kidney Disease (ADPKD) is a renal channelopathy due to loss-of-function mutations in the

Indexed as

ATPcalcimimeticscalcium-sensing receptorciPTECmitochondriarenal channelopathies

Identifiers

PMID30197885
PMCPMC6117232
OpenAlexW2888069295

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.