Evidence map›Paper›PMID 30159412›Full record

ReviewNon-coding RNA research2016

Regulation of DNA repair by non-coding miRNAs.

Venkateswaran Natarajan

Open access · goldAbstract readReview
In one paragraph

Review in Non-coding RNA research, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
0.3field-weighted citation impact, top 30% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 23 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Hippo Pathway in Regulating Drug Resistance of Glioblastoma.International journal of molecular sciences · 2021
    Review
  6. Review
  7. Article
  8. Article
  9. Deregulated MicroRNA Signature Following Glioblastoma Irradiation.Cancer control : journal of the Moffitt Cancer Center
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author at 1 institution in 1 country.

Venkateswaran NatarajanDiagnostic Molecular Oncology Centre, Department of Pathology, National University Hospital, Singapore.
National University Hospital · SG

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

DNA repair is an important signaling mechanism that is necessary to maintain genomic stability. Various types of DNA repair proteins are involved in the repair of different types of DNA damage. However, most of the DNA repair proteins are modified post-translation in order to activate their repair function, such as, ubiquitination, phosphorylation, acetylation, etc. Similarly, DNA repair proteins are also regulated by posttranscriptional modifications. Non-coding microRNAs (miRNAs) induced posttranscriptional regulation of mRNAs has gained attention in recent years. MiRNA-induced regulation of DNA repair proteins is of great interest, owing to its potential role in cancer therapy. In this review, we have summarized the role of different miRNAs in the regulation of various types of DNA repair proteins, which are essential for the maintenance of genomic stability.

Indexed as

ATM, ataxia-telangiectasia mutatedATR, ataxia-telangiectasia mutated relatedBER, base excision repairDNA damageDNA repairDSB, double strand breakDSB repairFA, Fanconi anemiaGenomic instabilityHR, homologous recombinationmiRNAsMIS, micro-instability syndromeNERNER, nucleotide excision repairNHEJ, non-homologous end joiningTLS, translesion synthesis

Identifiers

PMID30159412
PMCPMC6096415
OpenAlexW2548957441

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.