Evidence map›Paper›PMID 30123132›Full record

ArticleFrontiers in pharmacology2018

Wei Wu, Xu Liu, Lumin Wei, Tong Li, Yi Zang, Yuting Qian, Tingting Bai, Juanjuan Li, Mingping Xie, Ying Zhu and 2 more

Open access · goldAbstract read
In one paragraph

Article in Frontiers in pharmacology, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
0.9field-weighted citation impact, top 26% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 16 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. UCHL3 promotes aerobic glycolysis of pancreatic cancer through upregulating LDHA expression.Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National Cancer Institute of Mexico · 2021
    Article
  5. Article
  6. Article
  7. Article
  8. Article
  9. Article
  10. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 2 institutions in 1 country.

Wei WuDepartment of Gastroenterology, Ruijin Hospital Affiliated to The Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Xu LiuDepartment of Gastroenterology, Ruijin Hospital Affiliated to The Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Lumin WeiDepartment of Gastroenterology, Ruijin Hospital Affiliated to The Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Tong LiDepartment of Gastroenterology, Ruijin Hospital Affiliated to The Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Yi ZangDepartment of Gastroenterology, Ruijin Hospital Affiliated to The Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Yuting QianDepartment of Gastroenterology, Ruijin Hospital Affiliated to The Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Tingting BaiDepartment of Gastroenterology, Ruijin Hospital Affiliated to The Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Juanjuan LiDepartment of Gastroenterology, Ruijin Hospital Affiliated to The Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Mingping XieDepartment of Gastroenterology, Ruijin Hospital Affiliated to The Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Ying ZhuDepartment of Gastroenterology, Ruijin Hospital Affiliated to The Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Qi WangDepartment of Gastroenterology, Ruijin Hospital Affiliated to The Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Lifu WangDepartment of Gastroenterology, Ruijin Hospital Affiliated to The Shanghai Jiao Tong University School of Medicine, Shanghai, China.
Ruijin Hospital · CNShanghai Jiao Tong University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Wnt1 inducible signaling pathway protein-1 (WISP1) may play an important role in promoting carcinogenesis. However, the biological function and underlying mechanism of WISP1 in pancreatic carcinogenesis still remains enigmatic. In this study, immunochemistry staining showed that protein levels of WISP1 were more significantly upregulated in pancreatic ductal adenocarcinoma (PDAC) tissues with

Indexed as

carcinogenesispancreatic cancerSiah1Tp53 mutationWISP1

Identifiers

PMID30123132
PMCPMC6085464
OpenAlexW2886698797

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.