Evidence map›Paper›PMID 30104253›Full record

ArticleMolecular and cellular biology2018

E2F1 Mediates the Retinoic Acid-Induced Transcription of

Jo Hae Park, Kunsoo Rhee

Open access · bronzeAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Molecular and cellular biology, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact, top 93% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 0 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Jo Hae ParkDepartment of Biological Sciences, Seoul National University, Seoul, South Korea.
Kunsoo RheeDepartment of Biological Sciences, Seoul National University, Seoul, South Korea rheek@snu.ac.kr.
Seoul National University · KR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The involvement of cell division in cellular differentiation has long been accepted. Cell division may be required not only for the expansion of a differentiated cell population but also for the execution of differentiation processes. Nonetheless, knowledge regarding how specific differentiation processes are controlled in a cell division-dependent manner is far from complete. Here, we determined the involvement of cell division in neuronal differentiation. We initially confirmed that cell division is an essential event for the neuronal differentiation of P19 embryonic carcinoma cells. We investigated the induction mechanisms of

Indexed as

AnimalsCell DifferentiationCell DivisionCell Line, TumorE2F1 Transcription FactorEmbryonal Carcinoma Stem CellsHeLa CellsHomeodomain ProteinsHumansMiceNeuronsPromoter Regions, GeneticRepressor ProteinsTranscription, GeneticTretinoinE2f1 protein, mouseE2F1 Transcription FactorHomeodomain ProteinsRepressor ProteinsTretinoinTshz1 protein, mousecell divisionE2Fembryonic carcinoma cellneuronal differentiationTshz1

Identifiers

PMID30104253
PMCPMC6189455
OpenAlexW2887243134

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.