Evidence map›Paper›PMID 30093899›Full record

ArticleFrontiers in immunology2018

Yuli Cheng, Xing Zhu, Xiaohuan Wang, Qinghui Zhuang, Xu Huyan, Ximeng Sun, Jingjing Huang, Bin Zhan, Xinping Zhu

Open access · goldAbstract read
In one paragraph

Article in Frontiers in immunology, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 21 papers.

0numbers the graph read from it
0cells of the map it votes in
21citing papers in PubMed
5.5field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

21 citing papers in PubMed, 43 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Review
  6. Eosinophils in Rheumatoid Arthritis: A Multifaceted Role in the Pathogenesis of the Disease.Biocell : official journal of the Sociedades Latinoamericanas de Microscopia Electronica ... et. al · 2025
    Article
  7. International journal of molecular sciences · 2024
    Article
  8. Article
  9. Biochemistry and biophysics reports · 2023
    Article
  10. Food and waterborne parasitology · 2022
    Article
  11. Article
  12. Article
  13. Article
  14. Article
  15. Review
  16. Therapeutic Effect ofFrontiers in cell and developmental biology · 2021
    Article
  17. Frontiers in cellular and infection microbiology · 2021
    Article
  18. Article
  19. PreliminaryCells · 2020
    Article
  20. Frontiers in immunology · 2020
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 2 institutions in 2 countries.

Yuli ChengDepartment of Medical Microbiology and Parasitology, School of Basic Medical Sciences, Capital Medical University, Beijing, China.
Xing ZhuDepartment of Medical Microbiology and Parasitology, School of Basic Medical Sciences, Capital Medical University, Beijing, China.
Xiaohuan WangDepartment of Medical Microbiology and Parasitology, School of Basic Medical Sciences, Capital Medical University, Beijing, China.
Qinghui ZhuangDepartment of Medical Microbiology and Parasitology, School of Basic Medical Sciences, Capital Medical University, Beijing, China.
Xu HuyanDepartment of Medical Microbiology and Parasitology, School of Basic Medical Sciences, Capital Medical University, Beijing, China.
Ximeng SunDepartment of Medical Microbiology and Parasitology, School of Basic Medical Sciences, Capital Medical University, Beijing, China.
Jingjing HuangDepartment of Medical Microbiology and Parasitology, School of Basic Medical Sciences, Capital Medical University, Beijing, China.
Bin ZhanDepartment of Pediatrics, National School of Tropical Medicine, Baylor College of Medicine, Houston, TX, United States.
Xinping ZhuDepartment of Medical Microbiology and Parasitology, School of Basic Medical Sciences, Capital Medical University, Beijing, China.
Capital Medical University · CNBaylor College of Medicine · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Helminth infection induces Th2-biased immune responses and inhibitory/regulatory pathways that minimize excessive inflammation to facilitate the chronic infection of helminth in the host and in the meantime, prevent host hypersensitivity from autoimmune or atopic diseases. However, the detailed molecular mechanisms behind modulation on inflammatory diseases are yet to be clarified. Programmed death 1 (PD-1) is one of the important inhibitory receptors involved in the balance of host immune responses during chronic infection. Here, we used the murine model to examine the role of PD-1 in CD4

Indexed as

ImmunomodulationAnimalsArthritis, ExperimentalDisease Models, AnimalMiceProgrammed Cell Death 1 ReceptorT-Lymphocytopenia, Idiopathic CD4-PositiveTrichinella spiralisProgrammed Cell Death 1 ReceptorCD4+ T cellimmunomodulationprogrammed death 1rheumatoid arthritisTrichinella spiralis

Identifiers

PMID30093899
PMCPMC6070611
OpenAlexW2811442245

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.