ArticleToxicology research2016
Trichloroethylene-induced hypersensitivity dermatitis was associated with hepatic metabolic enzyme genes and immune-related genes.
Article in Toxicology research, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
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Who cites it
2 citing papers in PubMed, 5 citations in OpenAlex.
- HMGB 1 acetylation mediates trichloroethylene-induced immune kidney injury by facilitating endothelial cell-podocyte communication.Ecotoxicology and environmental safety · 2023Article
- Evaluation of Cytochrome P4502E1 mRNA Expression and Its Effects in Antioxidant Defenses, and Cell Toxicity in Printing Workers.Asian Pacific journal of cancer prevention : APJCP · 2022Article
Corrections and comments
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Authors and funding
9 authors at 2 institutions in 2 countries.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Trichloroethylene (TCE) is one of the common organic solvents that has been widely used in cleaning or degreasing of metal and electronic products. However, hundreds of cases of hypersensitivity dermatitis have occurred after the workers were occupationally exposed to TCE in China over the past decade. The purpose of this study was to investigate mRNA expression of hepatic metabolic enzyme genes, immune-related genes, apoptosis genes and oncogenes in patients with hypersensitivity dermatitis induced by trichloroethylene. 12 typical patients with TCE-induced hypersensitivity dermatitis were investigated as the study cases, peripheral blood samples were taken from patients and control, and real-time fluorescence PCR assay was applied for detection of mRNA expression of hepatic metabolic enzyme genes, immune-related genes, apoptosis genes and oncogenes. It was found that the relative levels of mRNA expression of
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Registered trials
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