Evidence map›Paper›PMID 29987222›Full record

ReviewMolecules (Basel, Switzerland)2018

Interleukin-33: Its Emerging Role in Allergic Diseases.

Wen Ding, Gui-Lin Zou, Wei Zhang, Xing-Ning Lai, Hou-Wen Chen, Li-Xia Xiong

Abstract readReview
In one paragraph

Review in Molecules (Basel, Switzerland), 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 28 papers.

0numbers the graph read from it
0cells of the map it votes in
28citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

28 citing papers in PubMed.

  1. Trial
  2. Experimental and therapeutic medicine · 2025
    Article
  3. Review
  4. Article
  5. Review
  6. Review
  7. Article
  8. Review
  9. Associations ofHeliyon · 2024
    Article
  10. Review
  11. Article
  12. Resolution Potential of Necrotic Cell Death Pathways.International journal of molecular sciences · 2022
    Review
  13. Article
  14. Review
  15. Article
  16. Review
  17. Article
  18. Article
  19. [Changes of serum interleukin-33 in preterm infants with bronchopulmonary dysplasia].Zhongguo dang dai er ke za zhi = Chinese journal of contemporary pediatrics · 2020
    Article
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Wen DingDepartment of Pathophysiology, Medical College, Nanchang University, 461 Bayi Road, Nanchang 330006, China. lmwl5@live.com.
Gui-Lin ZouDepartment of Pathophysiology, Medical College, Nanchang University, 461 Bayi Road, Nanchang 330006, China. z1293612123@gmail.com.
Wei ZhangGannan Medical University, Rongjiang New Area, Ganzhou 341000, China. zhangweign@outlook.com.
Xing-Ning LaiDepartment of Pathophysiology, Medical College, Nanchang University, 461 Bayi Road, Nanchang 330006, China. laixingning99@outlook.com.
Hou-Wen ChenDepartment of Pathophysiology, Medical College, Nanchang University, 461 Bayi Road, Nanchang 330006, China. chenhouwen@sina.com.
Li-Xia XiongDepartment of Pathophysiology, Medical College, Nanchang University, 461 Bayi Road, Nanchang 330006, China. xionglixia@ncu.edu.cn.ORCID 0000-0003-3769-2496

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Allergic diseases, which include asthma, allergic rhinitis (AR), chronic rhinosinusitis (CRS), atopic dermatitis (AD), food allergy (FA), allergic keratoconjunctivitis, seriously affect the quality of life of people all over the world. Recently, interleukin-33 (IL-33) has been found to play an important role in these refractory disorders, mainly by inducing T helper (Th) 2 immune responses. This article reviews the mobilization and biological function of IL-33 in allergic disorders, providing novel insights for addressing these hypersensitive conditions.

Indexed as

Up-RegulationHumansHypersensitivityInterleukin-1 Receptor-Like 1 ProteinInterleukin-33Quality of LifeSignal TransductionTh2 CellsIL1RL1 protein, humanIL33 protein, humanInterleukin-1 Receptor-Like 1 ProteinInterleukin-33allergic diseasesasthmainterleukin-33target therapyTh2-type immunity

Identifiers

PMID29987222
PMCPMC6099536

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.