Evidence map›Paper›PMID 29971205›Full record

ReviewKidney research and clinical practice2018

Pathology identifies glomerular treatment targets in diabetic nephropathy.

Charles E Alpers, Kelly L Hudkins

Abstract readReview
In one paragraph

Review in Kidney research and clinical practice, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed.

  1. Review
  2. Article
  3. Article
  4. Article
  5. Article
  6. Review
  7. Podocytes and microRNA-30/Cx43 axis in diabetic nephropathy.Annals of translational medicine · 2021
    Article
  8. Article
  9. Paclitaxel Ameliorates Palmitate-Induced Injury in Mouse Podocytes.Medical science monitor basic research · 2020
    Article
  10. Article
  11. The Journal of international medical research · 2020
    Article
  12. Review
  13. Adiponectin for the treatment of diabetic nephropathy.The Korean journal of internal medicine · 2019
    Review
  14. Article
  15. [NFAT2 mediates high glucose-induced apoptosis in glomerular podocytesNan fang yi ke da xue xue bao = Journal of Southern Medical University · 2018
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Charles E AlpersDepartment of Pathology, University of Washington, Seattle, WA, USA.
Kelly L HudkinsDepartment of Pathology, University of Washington, Seattle, WA, USA.

Funding

Vector and Transgenic Mouse CoreP30DK017047 · NIDDK · UNIVERSITY OF WASHINGTON · PI Sakeneh Zraika · 1986 to 2026
$41.4M
NIDDK NIH HHS P30 DK017047
6 · The paper itself

Abstract

The development of the glomerular injury in diabetic nephropathy involves interactions between podocytes, endothelium, and the mesangium. Loss of podocytes is an early and critical step in the development of diabetic nephropathy, and analysis of structural lesions within the mesangium such as mesangiolysis implicate the loss of podocytes as a key mediating event. The BTBR

Indexed as

Diabetic nephropathiesHistologyPathologyPodocytes

Identifiers

PMID29971205
PMCPMC6027807

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.