Evidence map›Paper›PMID 29928400›Full record

ArticleOncology letters2018

Genetic analysis and phosphoinositide 3-kinase/protein kinase B signaling pathway status in ovarian endometrioid borderline tumor samples.

Kohei Nakamura, Kentaro Nakayama, Masako Ishikawa, Toshiko Minamoto, Tomoka Ishibashi, Emi Sato, Kaori Sanuki, Hitomi Yamashita, Ruriko Ono, Kouji Iida and 4 more

Open access · diamondAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Oncology letters, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact, top 94% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 1 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors at 1 institution in 1 country.

Kohei NakamuraDepartment of Obstetrics and Gynecology, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan.
Kentaro NakayamaDepartment of Obstetrics and Gynecology, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan.
Masako IshikawaDepartment of Obstetrics and Gynecology, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan.
Toshiko MinamotoDepartment of Obstetrics and Gynecology, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan.
Tomoka IshibashiDepartment of Obstetrics and Gynecology, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan.
Emi SatoDepartment of Obstetrics and Gynecology, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan.
Kaori SanukiDepartment of Obstetrics and Gynecology, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan.
Hitomi YamashitaDepartment of Obstetrics and Gynecology, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan.
Ruriko OnoDepartment of Obstetrics and Gynecology, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan.
Kouji IidaDepartment of Obstetrics and Gynecology, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan.
Razia SultanaDepartment of Obstetrics and Gynecology, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan.
Mohammad Mahmud HossainDepartment of Obstetrics and Gynecology, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan.
Noriyoshi IshikawaDepartment of Organ Pathology, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan.
Satoru KyoDepartment of Obstetrics and Gynecology, Shimane University School of Medicine, Izumo, Shimane 693-8501, Japan.
Shimane University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Ovarian endometrioid borderline tumors (EBTs) are extremely rare, and are thought to be precursors of endometrioid carcinoma, beginning as adenofibroma or endometriosis and progressing in a slow, stepwise manner. In endometrioid carcinomas, a high frequency of activating mutations in phosphatase and tensin homolog (

Indexed as

AT-rich interaction domain 1Aendometrioid borderline tumormutationphosphatase and tensin homologphosphoinositide 3-kinase/protein kinase B signaling pathway

Identifiers

PMID29928400
PMCPMC6006484
OpenAlexW2801704792

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.