Evidence map›Paper›PMID 29798891›Full record

ArticleThe Journal of neuroscience : the official journal of the Society for Neuroscience2018

Lrfn2-Mutant Mice Display Suppressed Synaptic Plasticity and Inhibitory Synapse Development and Abnormal Social Communication and Startle Response.

Yan Li, Ryunhee Kim, Yi Sul Cho, Woo Seok Song, Doyoun Kim, Kyungdeok Kim, Junyeop Daniel Roh, Changuk Chung, Hanwool Park, Esther Yang and 6 more

Open access · bronzeAbstract read
In one paragraph

Article in The Journal of neuroscience : the official journal of the Society for Neuroscience, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 19 papers.

0numbers the graph read from it
0cells of the map it votes in
19citing papers in PubMed
1.5field-weighted citation impact, top 20% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

19 citing papers in PubMed, 30 citations in OpenAlex.

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  10. The Role of Zinc and NMDA Receptors in Autism Spectrum Disorders.Pharmaceuticals (Basel, Switzerland) · 2022
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

16 authors at 6 institutions in 2 countries.

Yan LiCenter for Synaptic Brain Dysfunctions, Institute for Basic Science (IBS), Daejeon 34141, Korea.
Ryunhee KimDepartment of Biological Sciences, Korea Advanced Institute of Science and Technology (KAIST), Daejeon 34141, Korea.ORCID 0000-0002-8206-6155
Yi Sul ChoDepartment of Anatomy and Neurobiology, School of Dentistry, Kyungpook National University, Daegu 700-412, Korea.ORCID 0000-0002-6310-8655
Woo Seok SongDepartment of Physiology, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.ORCID 0000-0002-8972-7526
Doyoun KimCenter for Synaptic Brain Dysfunctions, Institute for Basic Science (IBS), Daejeon 34141, Korea.
Kyungdeok KimDepartment of Biological Sciences, Korea Advanced Institute of Science and Technology (KAIST), Daejeon 34141, Korea.
Junyeop Daniel RohDepartment of Biological Sciences, Korea Advanced Institute of Science and Technology (KAIST), Daejeon 34141, Korea.
Changuk ChungCenter for Synaptic Brain Dysfunctions, Institute for Basic Science (IBS), Daejeon 34141, Korea.ORCID 0000-0003-3628-6090
Hanwool ParkGraduate School of Medical Science and Engineering, KAIST, Daejeon 34141, Korea.
Esther YangDepartment of Anatomy and Division of Brain Korea 21, Biomedical Science, College of Medicine, Korea University, Seoul 136-705, Korea, and.
Soo-Jeong KimDepartment of Brain and Cognitive Sciences, Daegu Gyeongbuk Institute of Science and Technology (DGIST), Daegu 42988, Korea.
Jaewon KoDepartment of Brain and Cognitive Sciences, Daegu Gyeongbuk Institute of Science and Technology (DGIST), Daegu 42988, Korea.ORCID 0000-0002-9117-4887
Hyun KimDepartment of Anatomy and Division of Brain Korea 21, Biomedical Science, College of Medicine, Korea University, Seoul 136-705, Korea, and.
Myoung-Hwan KimDepartment of Physiology, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.
Yong-Chul BaeDepartment of Anatomy and Neurobiology, School of Dentistry, Kyungpook National University, Daegu 700-412, Korea.ORCID 0000-0002-7618-418X
Eunjoon KimCenter for Synaptic Brain Dysfunctions, Institute for Basic Science (IBS), Daejeon 34141, Korea, kime@kaist.ac.kr.ORCID 0000-0001-5518-6584
Korea Advanced Institute of Science and Technology · KRInstitute for Basic Science · KRDaegu Gyeongbuk Institute of Science and Technology · KRKorea University · KRKyungpook National University · KRSeoul National University · KR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

SALM1 (SALM (synaptic adhesion-like molecule), also known as LRFN2 (leucine rich repeat and fibronectin type III domain containing), is a postsynaptic density (PSD)-95-interacting synaptic adhesion molecule implicated in the regulation of NMDA receptor (NMDAR) clustering largely based on

Indexed as

AnimalsMaleMembrane GlycoproteinsMiceMice, Inbred C57BLMice, KnockoutNerve Tissue ProteinsNeuronal PlasticityPyramidal CellsReflex, StartleSocial BehaviorSynapsesSynaptic TransmissionVocalization, AnimalLrfn2 protein, mouseMembrane GlycoproteinsNerve Tissue Proteinsexcitatory synaptic functionGABAergic neuronsinhibitory synapsesLrfn2NMDA receptorsocial communication

Identifiers

PMID29798891
PMCPMC6595971
OpenAlexW2788834053

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.