Evidence map›Paper›PMID 29769837›Full record

ReviewMediators of inflammation2018

The Role of Extracellular Adenosine Generation in the Development of Autoimmune Diseases.

F Morandi, A L Horenstein, R Rizzo, F Malavasi

Open access · goldAbstract readReview
In one paragraph

Review in Mediators of inflammation, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 30 papers.

0numbers the graph read from it
0cells of the map it votes in
30citing papers in PubMed
3.7field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

30 citing papers in PubMed, 47 citations in OpenAlex.

  1. AMetabolites · 2026
    Article
  2. Article
  3. Article
  4. The Role of Adenosine in Overcoming Resistance in Sarcomas.International journal of molecular sciences · 2024
    Review
  5. Article
  6. Article
  7. Adenosine AFrontiers in pharmacology · 2024
    Article
  8. Article
  9. Article
  10. Review
  11. Article
  12. How Does Vitamin D Affect Immune Cells Crosstalk in Autoimmune Diseases?International journal of molecular sciences · 2023
    Review
  13. Article
  14. Article
  15. Article
  16. Generation of a Retargeted OncolyticInternational journal of molecular sciences · 2021
    Article
  17. Review
  18. Inflammatory signaling mechanisms in bipolar disorder.Journal of biomedical science · 2021
    Review
  19. Downregulation of CD73/ABrain sciences · 2021
    Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 3 institutions in 1 country.

F MorandiStem Cell Laboratory and Cell Therapy Center, Istituto Giannina Gaslini, 16148 Genova, Italy.ORCID 0000-0002-2849-7595
A L HorensteinDepartment of Medical Sciences, Laboratory of Immunogenetics, University of Torino, 10126 Torino, Italy.ORCID 0000-0002-0382-0595
R RizzoDepartment of Medical Sciences, Section of Microbiology, University of Ferrara, 44121 Ferrara, Italy.
F MalavasiDepartment of Medical Sciences, Laboratory of Immunogenetics, University of Torino, 10126 Torino, Italy.
University of Turin · ITIstituto Giannina Gaslini · ITUniversity of Ferrara · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Adenosine (ADO) is an immunosuppressive molecule, which suppresses the immune responses by interacting with specific receptors expressed by immune effector cells. ADO is produced from ATP through the enzymatic activities of CD39 and CD73. Alternatively, ADO can be generated starting from NAD

Indexed as

5'-NucleotidaseAdenosineADP-ribosyl Cyclase 1AnimalsAntigens, CDApyraseAutoimmune DiseasesHumans5'-NucleotidaseAdenosineADP-ribosyl Cyclase 1Antigens, CDApyraseCD39 antigen

Identifiers

PMID29769837
PMCPMC5892213
OpenAlexW2790479524

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.