Evidence map›Paper›PMID 29740287›Full record

ArticleFrontiers in cellular neuroscience2018

Inhibition of GluR Current in Microvilli of Sensory Neurons via Na

Yasuhiro Kawasaki, Mitsuru Saito, Jonghwa Won, Jin Young Bae, Hajime Sato, Hiroki Toyoda, Eriko Kuramoto, Mikihiko Kogo, Takuma Tanaka, Takeshi Kaneko and 3 more

Open access · goldAbstract read
In one paragraph

Article in Frontiers in cellular neuroscience, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
0.1field-weighted citation impact, top 59% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 7 citations in OpenAlex.

  1. Article
  2. Review
  3. Review
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  5. Review
  6. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors at 5 institutions in 2 countries.

Yasuhiro KawasakiDepartment of Neuroscience and Oral Physiology, Graduate School of Dentistry, Osaka University, Osaka, Japan.
Mitsuru SaitoDepartment of Neuroscience and Oral Physiology, Graduate School of Dentistry, Osaka University, Osaka, Japan.
Jonghwa WonDepartment of Brain and Cognitive Sciences, College of Natural Sciences, Dental Research Institute and Department of Neurobiology and Physiology, School of Dentistry, Seoul National University, Seoul, South Korea.
Jin Young BaeDepartment of Oral Anatomy, School of Dentistry, Kyungpook National University, Daegu, South Korea.
Hajime SatoDepartment of Neuroscience and Oral Physiology, Graduate School of Dentistry, Osaka University, Osaka, Japan.
Hiroki ToyodaDepartment of Neuroscience and Oral Physiology, Graduate School of Dentistry, Osaka University, Osaka, Japan.
Eriko KuramotoDepartment of Morphological Brain Science, Graduate School of Medicine, Kyoto University, Kyoto, Japan.
Mikihiko KogoDepartment of Neuroscience and Oral Physiology, Graduate School of Dentistry, Osaka University, Osaka, Japan.
Takuma TanakaDepartment of Computational Intelligence and Systems Science, Interdisciplinary Graduate School of Science and Engineering, Tokyo Institute of Technology, Yokohama, Japan.
Takeshi KanekoDepartment of Morphological Brain Science, Graduate School of Medicine, Kyoto University, Kyoto, Japan.
Seog Bae OhDepartment of Brain and Cognitive Sciences, College of Natural Sciences, Dental Research Institute and Department of Neurobiology and Physiology, School of Dentistry, Seoul National University, Seoul, South Korea.
Yong Chul BaeDepartment of Oral Anatomy, School of Dentistry, Kyungpook National University, Daegu, South Korea.
Youngnam KangDepartment of Neuroscience and Oral Physiology, Graduate School of Dentistry, Osaka University, Osaka, Japan.
Osaka University · JPKyoto University · JPKyungpook National University · KRSeoul National University · KRTokyo Institute of Technology · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Glutamatergic dendritic EPSPs evoked in cortical pyramidal neurons are depressed by activation of hyperpolarization-activated cyclic nucleotide-gated (HCN) channels expressed in dendritic spines. This depression has been attributed to shunting effects of HCN current (

Indexed as

glutamate receptorhyperpolarization-activated cyclic nucleotide-gated cation channelmesencephalic trigeminal nucleusmicrovilliNa+/K+ pumpprimary sensory neuron

Identifiers

PMID29740287
PMCPMC5928758
OpenAlexW2802909692

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.