Evidence map›Paper›PMID 29670615›Full record

ArticleFrontiers in immunology2018

Distinct

Eszter Baricza, Nikolett Marton, Panna Királyhidi, Orsolya Tünde Kovács, Ilona Kovácsné Székely, Eszter Lajkó, Lászó Kőhidai, Bernadett Rojkovich, Barbara Érsek, Edit Irén Buzás and 1 more

Open access · goldAbstract readComparative Study
PubMed Full text at PMC Publisher
In one paragraph

Article in Frontiers in immunology, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
0.6field-weighted citation impact, top 36% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 13 citations in OpenAlex.

  1. Review
  2. Article
  3. From PsO to PsA: the role of TFrontiers in medicine · 2024
    Review
  4. Review
  5. Article
  6. Review
  7. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 3 institutions in 1 country.

Eszter BariczaDepartment of Genetics, Cell- and Immunobiology, Semmelweis University, Budapest, Hungary.
Nikolett MartonDepartment of Genetics, Cell- and Immunobiology, Semmelweis University, Budapest, Hungary.
Panna KirályhidiDepartment of Genetics, Cell- and Immunobiology, Semmelweis University, Budapest, Hungary.
Orsolya Tünde KovácsDepartment of Genetics, Cell- and Immunobiology, Semmelweis University, Budapest, Hungary.
Ilona Kovácsné SzékelyDepartment of Methodology, Budapest Business School, Budapest, Hungary.
Eszter LajkóDepartment of Genetics, Cell- and Immunobiology, Semmelweis University, Budapest, Hungary.
Lászó KőhidaiDepartment of Genetics, Cell- and Immunobiology, Semmelweis University, Budapest, Hungary.
Bernadett RojkovichBuda Hospital of the Hospitaller Order of Saint John of God, Budapest, Hungary.
Barbara ÉrsekDepartment of Genetics, Cell- and Immunobiology, Semmelweis University, Budapest, Hungary.
Edit Irén BuzásDepartment of Genetics, Cell- and Immunobiology, Semmelweis University, Budapest, Hungary.
György NagyDepartment of Genetics, Cell- and Immunobiology, Semmelweis University, Budapest, Hungary.
Semmelweis University · HUBuda Health Center · HUBudapest Business University · HU

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: The T-helper 17 (Th17) cells have a prominent role in inflammation as well as in bone and join destruction in both rheumatoid and psoriatic arthritis (RA and PsA). Here, we studied Th17 cell differentiation in RA and PsA. Methods: Blood samples from healthy donors, RA and PsA patients were collected. CD45RO Results: Conclusion: The naive CD4 T-lymphocytes are predisposed to differentiate into Th17 cells and the

Indexed as

Arthritis, PsoriaticArthritis, RheumatoidBlood CirculationCell DifferentiationCells, CulturedFlow CytometryHumansImmunityImmunologic MemoryInterleukin-22InterleukinsLeukocyte Common AntigensNuclear Receptor Subfamily 1, Group F, Member 3Receptors, CCR6T-bet Transcription FactorT-Box Domain ProteinsCCR6 protein, humanInterleukin-22InterleukinsLeukocyte Common AntigensNuclear Receptor Subfamily 1, Group F, Member 3Receptors, CCR6RORC protein, humanT-bet Transcription FactorT-Box Domain Proteinsinterleukin-17Ainterleukin-22psoriatic arthritisrheumatoid arthritisT-helper 17 differentiation

Identifiers

PMID29670615
PMCPMC5893718
OpenAlexW2795419427

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.