Evidence map›Paper›PMID 29666621›Full record

ArticleFrontiers in immunology2018

The Role of AIRE in the Immunity Against

Jose Antonio Tavares de Albuquerque, Pinaki Prosad Banerjee, Angela Castoldi, Royce Ma, Nuria Bengala Zurro, Leandro Hideki Ynoue, Christina Arslanian, Marina Uchoa Wall Barbosa-Carvalho, Joya Emilie de Menezes Correia-Deur, Fernanda Guimarães Weiler and 7 more

Open access · goldAbstract read
In one paragraph

Article in Frontiers in immunology, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
1.3field-weighted citation impact, top 21% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 13 citations in OpenAlex.

  1. Review
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  6. Pathogens (Basel, Switzerland) · 2019
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

17 authors at 5 institutions in 3 countries.

Jose Antonio Tavares de AlbuquerqueDepartment of Immunology, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil.
Pinaki Prosad BanerjeeCenter for Human Immunobiology, Texas Children's Hospital, Houston, TX, United States.
Angela CastoldiDepartment of Immunology, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil.
Royce MaCenter for Human Immunobiology, Texas Children's Hospital, Houston, TX, United States.
Nuria Bengala ZurroDepartment of Immunology, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil.
Leandro Hideki YnoueDepartment of Immunology, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil.
Christina ArslanianDepartment of Immunology, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil.
Marina Uchoa Wall Barbosa-CarvalhoDepartment of Immunology, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil.
Joya Emilie de Menezes Correia-DeurEscola Paulista de Medicina, Universidade Federal de São Paulo, São Paulo, Brazil.
Fernanda Guimarães WeilerEscola Paulista de Medicina, Universidade Federal de São Paulo, São Paulo, Brazil.
Magnus Regios Dias-da-SilvaEscola Paulista de Medicina, Universidade Federal de São Paulo, São Paulo, Brazil.
Marise Lazaretti-CastroEscola Paulista de Medicina, Universidade Federal de São Paulo, São Paulo, Brazil.
Luis Alberto PedrozaColegio de Ciencias de la Salud, Escuela de Medicina, Hospital de los Valles, Universidad San Francisco de Quito, Quito, Ecuador.
Niels Olsen Saraiva CâmaraDepartment of Immunology, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil.
Emily MaceCenter for Human Immunobiology, Texas Children's Hospital, Houston, TX, United States.
Jordan Scott OrangeCenter for Human Immunobiology, Texas Children's Hospital, Houston, TX, United States.
Antonio Condino-NetoDepartment of Immunology, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil.
Universidade de São Paulo · BRUniversidade Federal de São Paulo · BRBaylor College of Medicine · USTexas Children's Hospital · USUniversidad San Francisco de Quito · EC

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Autoimmune-polyendocrinopathy-candidiasis-ectodermal dystrophy (APECED) is a primary immunodeficiency caused by mutations in the autoimmune regulator gene (

Indexed as

AIRE ProteinCandida albicansCandidiasisCytokinesDectin-1HEK293 CellsHumansHyphaeLectins, C-TypeMacrophagesMutationPhagocytosisPolyendocrinopathies, AutoimmuneRNA InterferenceTHP-1 CellsTranscription FactorsAIRE ProteinCLEC6A protein, humanCytokinesDectin-1Lectins, C-TypeTranscription FactorsAIREAPECEDC. albicansDectin receptorhyphaemacrophagesreceptor recruitment

Identifiers

PMID29666621
PMCPMC5875531
OpenAlexW2793954246

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.