Evidence map›Paper›PMID 29637414›Full record

ReviewCurrent rheumatology reports2018

Dermatomyositis Clinical and Pathological Phenotypes Associated with Myositis-Specific Autoantibodies.

Paige W Wolstencroft, David F Fiorentino

Abstract readReview
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In one paragraph

Review in Current rheumatology reports, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 54 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
54citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

54 citing papers in PubMed, 1 synthesis or guideline pooled it.

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  17. Research progress on the dermatomyositis specific autoantibodies and malignancy associated dermatomyositis.Zhong nan da xue xue bao. Yi xue ban = Journal of Central South University. Medical sciences · 2023
    Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Paige W WolstencroftDepartment of Dermatology, Stanford University School of Medicine, 450 Broadway Street, Redwood City, CA, 94063, USA.
David F FiorentinoDepartment of Dermatology, Stanford University School of Medicine, 450 Broadway Street, Redwood City, CA, 94063, USA. fiorentino@stanford.edu.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

purpose of reviewDermatomyositis is an idiopathic inflammatory myopathy with a variety of systemic and cutaneous manifestations. The myositis-specific autoantibodies (MSAs) are associated with phenotypic features and provide a tool for sub-classification of dermatomyositis patients. This review focuses on recent work characterizing the clinical features that accompany the MSAs in dermatomyositis. RECENT

findingsThere is increasing recognition of the distinct clinical and pathological phenotypes associated with each MSA. Most of these features display considerable overlap between MSA groups. Despite this, there are notable differences between the typical combinations of cutaneous and systemic manifestations, response to therapy, prognosis, and disease sequelae that define each dermatomyositis MSA group. The MSAs may ultimately improve diagnosis and sub-classification of dermatomyositis patients. However, more work is needed to understand the pathologic basis for much of the heterogeneity found within these subgroups.

Indexed as

Adenosine TriphosphatasesAutoantibodiesBiomarkersDermatomyositisDNA-Binding ProteinsHumansInterferon-Induced Helicase, IFIH1Mi-2 Nucleosome Remodeling and Deacetylase ComplexPhenotypePrognosisTranscription FactorsUbiquitin-Activating EnzymesAdenosine TriphosphatasesAutoantibodiesBiomarkersCHD4 protein, humanDNA-Binding ProteinsIFIH1 protein, humanInterferon-Induced Helicase, IFIH1Mi-2 Nucleosome Remodeling and Deacetylase ComplexMORC3 protein, humanSAE1 protein, humanTranscription FactorsTRIM33 protein, humanUbiquitin-Activating EnzymesAutoantibodiesAutoimmune diseaseDermatomyositisMSAsMyositis-specific autoantibodies

Identifiers

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.