Evidence map›Paper›PMID 29602972›Full record

ArticleParasitology research2018

The potential role of toll-like receptor 4 Asp299Gly polymorphism and its association with recurrent cystic echinococcosis in postoperative patients.

Jafar Noori, Adel Spotin, Ehsan Ahmadpour, Mahmoud Mahami-Oskouei, Homayoun Sadeghi-Bazargani, Tohid Kazemi, Ebrahim Sakhinia, Leili Aghebati-Maleki, Firooz Shahrivar

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Article in Parasitology research, 2018. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
0.8field-weighted citation impact, top 28% of its field
1 · What the graph read from it

What it found

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2 · The registry

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3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 13 citations in OpenAlex.

  1. Article
  2. Host genetic backgrounds: the key to determining parasite-host adaptation.Frontiers in cellular and infection microbiology · 2023
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4 · The record

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5 · Who and what money

Authors and funding

9 authors at 1 institution in 1 country.

Jafar NooriImmunology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
Adel SpotinImmunology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran. Adelespotin@gmail.com.
Ehsan AhmadpourInfectious and Tropical Diseases Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
Mahmoud Mahami-OskoueiDepartment of Parasitology and Mycology, Faculty of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran.
Homayoun Sadeghi-BazarganiRoad Traffic Injury Research Centre, Tabriz University of Medical Sciences, Tabriz, Iran.
Tohid KazemiImmunology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
Ebrahim SakhiniaDepartment of Biochemistry, Faculty of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran.
Leili Aghebati-MalekiImmunology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
Firooz ShahrivarDepartment of Parasitology and Mycology, Faculty of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran.
Tabriz University of Medical Sciences · IR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The study of pathogenesis mechanisms of larval stages in the Taeniidae has recently focused on host genetic factors, particularly toll-like receptor (TLR) variations. However, the potential role of TLR4 polymorphism in hydatidosis has not yet been sufficiently elucidated in postoperative patients. In this case-control investigation, 80 patients from Iran, including 40 with acute hydatidosis (AH) and 40 with recurrent hydatidosis (RH), and 80 ethnically matched controls were evaluated from February 2015 to February 2017. Hydatidosis patients were confirmed using radiological, immunological, and histopathological examinations. Genotyping of Asp299Gly and Thr399Ile of TLR4 single-nucleotide polymorphisms was determined by restriction fragment length polymorphism, sequencing, and phylogenetic strategies. The heterozygous mutant-type TLR4 Asp299Gly genotype indicated a tendency to be associated with the occurrence of RH (P = 0.060) and conferred a 3-fold risk for susceptibility. There was no difference in genotype frequency of Asp299Gly between patients with AH and healthy controls (P = 0.42; OR, 1.82; 95% CI, 0.11-30.1%). Interestingly, a frequency of the G allele (12%: Gly) was observed to be a risk factor for susceptibility to RH patients (P = 0.050; OR, 7.08; 95% CI, 0.97-51.5%). A relative genetic variability of TLR4 Asp299Gly was found in RH patients (haplotype diversity: 0.700) compared to AH patients and healthy controls (Hd: 0.000). The Asp299Gly genotype was dominantly identified in patients with hepatic hydatid cysts. The TLR4 Thr399Ile codon was not detected except in a patient with a pulmonary hydatid cyst. The current findings enhance our knowledge regarding the TLR4 Asp299Gly polymorphism potentially leading to the development of RH, by skewing the immune system towards a Th2 response. Identification of the Asp299Gly codon may be a diagnostic hallmark in RH patients who have undergone unsuccessful postoperative intervention. However, further studies with a higher case number are needed on ethnic population from various geographic regions, in order to confirm this hypothesis.

Indexed as

AdultAllelesAnimalsCase-Control StudiesEchinococcosisEchinococcus granulosusFemaleGenetic Predisposition to DiseaseGenotypeHaplotypesHumansIranMaleMiddle AgedPhylogenyPolymorphism, Restriction Fragment LengthTLR4 protein, humanToll-Like Receptor 4Innate immunityRecurrent hydatidosisToll-like receptor 4Unsuccessful postoperative intervention

Identifiers

PMID29602972
OpenAlexW2795227553

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.